Multiple virulence factors are required for Staphylococcus aureus-induced apoptosis in endothelial cells.
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A global view of Staphylococcus aureus whole genome expression upon internalization in human epithelial cellsDifferential responses of osteoblasts and macrophages upon Staphylococcus aureus infectionSigma Factor SigB Is Crucial to Mediate Staphylococcus aureus Adaptation during Chronic InfectionsDivergent responses of different endothelial cell types to infection with Candida albicans and Staphylococcus aureusNatural mutations in a Staphylococcus aureus virulence regulator attenuate cytotoxicity but permit bacteremia and abscess formationMechanisms of infective endocarditis: pathogen-host interaction and risk states.Staphylococcal alpha-toxin is not sufficient to mediate escape from phagolysosomes in upper-airway epithelial cells.Bacteria tracking by in vivo magnetic resonance imaging.PSMs of hypervirulent Staphylococcus aureus act as intracellular toxins that kill infected osteoblasts.A potential new pathway for Staphylococcus aureus dissemination: the silent survival of S. aureus phagocytosed by human monocyte-derived macrophages.Stimulation of platelet apoptosis by peptidoglycan from Staphylococcus aureus 113.Staphylococcal PknB as the first prokaryotic representative of the proline-directed kinases.Two novel point mutations in clinical Staphylococcus aureus reduce linezolid susceptibility and switch on the stringent response to promote persistent infection.Genome wide association study of SNP-, gene-, and pathway-based approaches to identify genes influencing susceptibility to Staphylococcus aureus infections.Identification of single nucleotide polymorphisms associated with hyperproduction of alpha-toxin in Staphylococcus aureus.Staphylococcus aureus protein A binds to osteoblasts and triggers signals that weaken bone in osteomyelitis.Inactivation of thyA in Staphylococcus aureus attenuates virulence and has a strong impact on metabolism and virulence gene expression.In vitro characterization of representative clinical South African Staphylococcus aureus isolates from various clonal lineagesIntracellular Staphylococcus aureus: the Trojan horse of recalcitrant chronic rhinosinusitis?Secretome analysis defines the major role of SecDF in Staphylococcus aureus virulenceSigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variantsTherapy of intracellular Staphylococcus aureus by tigecyclin.Complete Genome Sequence of Staphylococcus aureus 6850, a Highly Cytotoxic and Clinically Virulent Methicillin-Sensitive Strain with Distant Relatedness to Prototype Strains.Proteomic and genomic analysis of methicillin-resistant Staphylococcus aureus (MRSA) exposed to manuka honey in vitro demonstrated down-regulation of virulence markersIn vitro endothelial cell damage is positively correlated with enhanced virulence and poor vancomycin responsiveness in experimental endocarditis due to methicillin-resistant Staphylococcus aureus.Staphylococcus aureus induces hypoxia and cellular damage in porcine dermal explantsStaphylococcus aureus phenotype switching: an effective bacterial strategy to escape host immune response and establish a chronic infectionStaphylococcus aureus Panton-Valentine leukocidin induces an inflammatory response in human phagocytes via the NLRP3 inflammasome.Lex marks the spot: the virulent side of SOS and a closer look at the LexA regulon.Genome content determination in methicillin-resistant Staphylococcus aureus.Differential response of bovine mammary epithelial cells to Staphylococcus aureus or Escherichia coli agonists of the innate immune system.Role of SCCmec type in outcome of Staphylococcus aureus bacteremia in a single medical center.Functional characterization of the sigmaB-dependent yabJ-spoVG operon in Staphylococcus aureus: role in methicillin and glycopeptide resistance.Cytoplasmic replication of Staphylococcus aureus upon phagosomal escape triggered by phenol-soluble modulin α.Staphylococcus aureus adaptation to the host and persistence: role of loss of capsular polysaccharide expression.Intracellular Staphylococcus aureus: live-in and let die.Influence of the Paracoccidioides brasiliensis 14-3-3 and gp43 proteins on the induction of apoptosis in A549 epithelial cells.Staphylococcus aureus-Associated Musculoskeletal Infections.Staphylococcus haemolyticus strains target mitochondria and induce caspase-dependent apoptosis of macrophages.Expression of δ-toxin by Staphylococcus aureus mediates escape from phago-endosomes of human epithelial and endothelial cells in the presence of β-toxin.
P2860
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P2860
Multiple virulence factors are required for Staphylococcus aureus-induced apoptosis in endothelial cells.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Multiple virulence factors are ...... poptosis in endothelial cells.
@en
Multiple virulence factors are ...... poptosis in endothelial cells.
@nl
type
label
Multiple virulence factors are ...... poptosis in endothelial cells.
@en
Multiple virulence factors are ...... poptosis in endothelial cells.
@nl
prefLabel
Multiple virulence factors are ...... poptosis in endothelial cells.
@en
Multiple virulence factors are ...... poptosis in endothelial cells.
@nl
P2093
P2860
P1476
Multiple virulence factors are ...... poptosis in endothelial cells.
@en
P2093
Ambrose L Cheung
Bettina Haslinger-Löffler
Bhanu Sinha
Britta Wagner
Georg Peters
Katrin Strangfeld
Klaus Schulze-Osthoff
Matthias Grundmeier
Ute Fischer
P2860
P304
P356
10.1111/J.1462-5822.2005.00533.X
P577
2005-08-01T00:00:00Z