Glutamate release by an Na+ load and oxidative stress in nerve terminals: relevance to ischemia/reperfusion.
about
HDT-1, a new synthetic compound, inhibits glutamate release in rat cerebral cortex nerve terminals (synaptosomes).Glycine release provoked by disturbed Na⁺, Na⁺ and Ca²⁺ homeostasis in cerebellar nerve endings: roles of Ca²⁺ channels, Na⁺/Ca²⁺ exchangers and GlyT2 transporter reversal.HTDP-2, a new synthetic compound, inhibits glutamate release through reduction of voltage-dependent Ca²⁺ influx in rat cerebral cortex nerve terminals.
P2860
Glutamate release by an Na+ load and oxidative stress in nerve terminals: relevance to ischemia/reperfusion.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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name
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@en
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@nl
type
label
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@en
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@nl
prefLabel
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@en
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@nl
P2860
P1476
Glutamate release by an Na+ lo ...... vance to ischemia/reperfusion.
@en
P2093
Laszlo Tretter
P2860
P304
P356
10.1046/J.1471-4159.2002.01191.X
P407
P577
2002-11-01T00:00:00Z