Microglia enhance beta-amyloid peptide-induced toxicity in cortical and mesencephalic neurons by producing reactive oxygen species.
about
Imaging of peripheral benzodiazepine receptor expression as biomarkers of detrimental versus beneficial glial responses in mouse models of Alzheimer's and other CNS pathologiesParkinson's disease and exposure to infectious agents and pesticides and the occurrence of brain injuries: role of neuroinflammation.Role of ROS and RNS Sources in Physiological and Pathological ConditionsProteomics in Traditional Chinese Medicine with an Emphasis on Alzheimer's DiseaseFriends or Foes: Matrix Metalloproteinases and Their Multifaceted Roles in Neurodegenerative DiseasesMicroglial cell dysregulation in brain aging and neurodegenerationProgress towards biocompatible intracortical microelectrodes for neural interfacing applicationsChronic apocynin treatment attenuates beta amyloid plaque size and microglial number in hAPP(751)(SL) miceEndogenous dynorphin protects against neurotoxin-elicited nigrostriatal dopaminergic neuron damage and motor deficits in miceNADPH oxidase as a therapeutic target in Alzheimer's disease.Fibrillar beta-amyloid peptide Abeta1-40 activates microglial proliferation via stimulating TNF-alpha release and H2O2 derived from NADPH oxidase: a cell culture studyLow energy laser light (632.8 nm) suppresses amyloid-β peptide-induced oxidative and inflammatory responses in astrocytes.Reactive microgliosis: extracellular micro-calpain and microglia-mediated dopaminergic neurotoxicity.WNT5A signaling contributes to Aβ-induced neuroinflammation and neurotoxicity.Role of microglia adenosine A(2A) receptors in retinal and brain neurodegenerative diseases.Role of microglia in neurotrauma.Critical role of NADPH oxidase in neuronal oxidative damage and microglia activation following traumatic brain injury.Transcriptome analysis of amoeboid and ramified microglia isolated from the corpus callosum of rat brain.Low-level laser therapy regulates microglial function through Src-mediated signaling pathways: implications for neurodegenerative diseasesMicroglial MAC1 receptor and PI3K are essential in mediating β-amyloid peptide-induced microglial activation and subsequent neurotoxicityMicroglial P2X₇ receptor expression is accompanied by neuronal damage in the cerebral cortex of the APPswe/PS1dE9 mouse model of Alzheimer's disease.sym-Triazines for directed multitarget modulation of cholinesterases and amyloid-β in Alzheimer's disease.Clozapine protects dopaminergic neurons from inflammation-induced damage by inhibiting microglial overactivation.Prolonged minocycline treatment impairs motor neuronal survival and glial function in organotypic rat spinal cord cultures.Anti-inflammatory effects of BHBA in both in vivo and in vitro Parkinson's disease models are mediated by GPR109A-dependent mechanisms.Microglia lacking E Prostanoid Receptor subtype 2 have enhanced Abeta phagocytosis yet lack Abeta-activated neurotoxicity.The microglial NADPH oxidase complex as a source of oxidative stress in Alzheimer's diseaseCattle encephalon glycoside and ignotin injection improves cognitive impairment in APPswe/PS1dE9 mice used as multitarget anti-Alzheimer's drug candidatesA novel role of microglial NADPH oxidase in mediating extra-synaptic function of norepinephrine in regulating brain immune homeostasis.Cerebrometabolic abnormalities in Alzheimer's disease.Regulation of cyclic AMP response element-binding protein during neuroglial interactions.Reactive oxygen species and inhibitors of inflammatory enzymes, NADPH oxidase, and iNOS in experimental models of Parkinson's disease.BBB-Permeable, Neuroprotective, and Neurotrophic Polysaccharide, Midi-GAGRInhibition of NADPH oxidase promotes alternative and anti-inflammatory microglial activation during neuroinflammationLow-level laser therapy for beta amyloid toxicity in rat hippocampus.NADPH oxidases: novel therapeutic targets for neurodegenerative diseasesNeuroprotective properties of the innate immune system and bone marrow stem cells in Alzheimer's disease.Traumatic brain injury in aged animals increases lesion size and chronically alters microglial/macrophage classical and alternative activation statesMitochondrial oxidative damage in aging and Alzheimer's disease: implications for mitochondrially targeted antioxidant therapeuticsFormyl-methionyl-leucyl-phenylalanine-induced dopaminergic neurotoxicity via microglial activation: a mediator between peripheral infection and neurodegeneration?
P2860
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P2860
Microglia enhance beta-amyloid peptide-induced toxicity in cortical and mesencephalic neurons by producing reactive oxygen species.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@en
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@nl
type
label
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@en
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@nl
prefLabel
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@en
Microglia enhance beta-amyloid ...... ucing reactive oxygen species.
@nl
P2093
P1476
Microglia enhance beta-amyloid ...... ducing reactive oxygen species
@en
P2093
Belinda Wilson
Cynthia Cooper
P304
P356
10.1046/J.1471-4159.2002.01210.X
P407
P577
2002-11-01T00:00:00Z