about
A tissue-specific approach to the analysis of metabolic changes in Caenorhabditis elegansRelationship between mitochondrial electron transport chain dysfunction, development, and life extension in Caenorhabditis elegansGenetics of aging in Caenorhabditis elegansCaenorhabditis elegans as a Model System for Studying Drug Induced Mitochondrial ToxicityGenes that may modulate longevity in C. elegans in both dauer larvae and long-lived daf-2 adultsBridging the phenotypic gap: real-time assessment of mitochondrial function and metabolism of the nematode Caenorhabditis elegans.Dynamic regulation of genetic pathways and targets during aging in Caenorhabditis elegans.Disruption of insulin signalling preserves bioenergetic competence of mitochondria in ageing Caenorhabditis elegans.Uncoupling the pleiotropic phenotypes of clk-1 with tRNA missense suppressors in Caenorhabditis elegans.The lin-4 gene controls fat accumulation and longevity in Caenorhabditis elegansMitochondrial and cytoplasmic ROS have opposing effects on lifespan.Dietary restriction in the nematode Caenorhabditis elegans.A Measurable increase in oxidative damage due to reduction in superoxide detoxification fails to shorten the life span of long-lived mitochondrial mutants of Caenorhabditis elegansSecrets of the lac operon. Glucose hysteresis as a mechanism in dietary restriction, aging and disease.Rapid sublethal toxicity assessment using bioluminescent Caenorhabditis elegans, a novel whole-animal metabolic biosensorROS in aging Caenorhabditis elegans: damage or signaling?Decreased energy metabolism extends life span in Caenorhabditis elegans without reducing oxidative damage.Lifespan extension and paraquat resistance in a ubiquinone-deficient Escherichia coli mutant depend on transcription factors ArcA and TdcA.Mitochondrial oxidative phosphorylation is defective in the long-lived mutant clk-1.Cell Biology of the Mitochondrion.Molecular mechanism of maternal rescue in the clk-1 mutants of Caenorhabditis elegans.Lack of peroxisomal catalase causes a progeric phenotype in Caenorhabditis elegans.Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans.
P2860
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P2860
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
No reduction of energy metabolism in Clk mutants.
@en
No reduction of energy metabolism in Clk mutants.
@nl
type
label
No reduction of energy metabolism in Clk mutants.
@en
No reduction of energy metabolism in Clk mutants.
@nl
prefLabel
No reduction of energy metabolism in Clk mutants.
@en
No reduction of energy metabolism in Clk mutants.
@nl
P2093
P50
P1476
No reduction of energy metabolism in Clk mutants.
@en
P2093
Hilda Raes
Isabelle Lenaerts
Kristel Brys
Sylvie Van Eygen
P304
P356
10.1016/S0047-6374(02)00085-4
P577
2002-09-01T00:00:00Z