about
Interferon-alpha, immune activation and immune dysfunction in treated HIV infectionProduction of IgG antibodies to pneumococcal polysaccharides is associated with expansion of ICOS+ circulating memory T follicular-helper cells which is impaired by HIV infection.Impaired CTLA-4 responses in COPD are associated with systemic inflammationImmunosenescent CD57+CD4+ T-cells accumulate and contribute to interferon-γ responses in HIV patients responding stably to ART.Could a loss of memory T cells limit responses to hepatitis C virus (HCV) antigens in blood leucocytes from patients chronically infected with HCV before and during pegylated interferon-alpha and ribavirin therapy?Toll-like receptor (TLR) expression on CD4+ and CD8+ T-cells in patients chronically infected with hepatitis C virus.The proportion and function of peripheral myeloid-derived suppressor cells do not correlate with systemic inflammation in chronic obstructive pulmonary disease.Impaired function of regulatory T-cells in patients with chronic obstructive pulmonary disease (COPD).Thymic tissue is not evident on high-resolution computed tomography and [¹⁸F]fluoro-deoxy-glucose positron emission tomography scans of aviraemic HIV patients with poor recovery of CD4⁺ T cells.IFN-α exerts opposing effects on activation-induced and IL-7-induced proliferation of T cells that may impair homeostatic maintenance of CD4+ T cell numbers in treated HIV infection.CD31 (PECAM-1) is a marker of recent thymic emigrants among CD4+ T-cells, but not CD8+ T-cells or gammadelta T-cells, in HIV patients responding to ART.Thymic function in severely immunodeficient HIV type 1-infected patients receiving stable and effective antiretroviral therapy.Could natural killer cells compensate for impaired CD4+ T-cell responses to CMV in HIV patients responding to antiretroviral therapy?Circulating mycobacterial-reactive CD4+ T cells with an immunosuppressive phenotype are higher in active tuberculosis than latent tuberculosis infection.The number and function of circulating dendritic cells may limit effector memory CD4+ T-cell responses in HIV patients responding to antiretroviral therapy.Low CD4+ T-cell counts in HIV patients receiving effective antiretroviral therapy are associated with CD4+ T-cell activation and senescence but not with lower effector memory T-cell function.CD4+ T-Cell Deficiency in HIV Patients Responding to Antiretroviral Therapy Is Associated With Increased Expression of Interferon-Stimulated Genes in CD4+ T CellsVaccine-induced IgG2 anti-HIV p24 is associated with control of HIV in patients with a ‘high-affinity’ FcγRIIa genotypeInterleukin-7 signalling defects in naive CD4+ T cells of HIV patients with CD4+ T-cell deficiency on antiretroviral therapy are associated with T-cell activation and senescenceIsotype-switched immunoglobulin G antibodies to HIV Gag proteins may provide alternative or additional immune responses to ‘protective’ human leukocyte antigen-B alleles in HIV controllersChemokine receptor expression on dendritic cells is normal in HIV-infected patients with a stable response to art, but chemokine levels remain elevatedNadir CD4 T-Cell Counts Continue to Influence Interferon-γ Responses in HIV Patients Who Began Antiretroviral Treatment With Advanced ImmunodeficiencySuccessful treatment of macrophage activation syndrome complicating adult Still disease with anakinra
P50
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Sonia Fernandez
@ast
Sonia Fernandez
@en
Sonia Fernandez
@es
Sonia Fernandez
@nl
Sonia Fernandez
@sl
type
label
Sonia Fernandez
@ast
Sonia Fernandez
@en
Sonia Fernandez
@es
Sonia Fernandez
@nl
Sonia Fernandez
@sl
prefLabel
Sonia Fernandez
@ast
Sonia Fernandez
@en
Sonia Fernandez
@es
Sonia Fernandez
@nl
Sonia Fernandez
@sl
P1053
H-9176-2014
P106
P21
P31
P3829
P496
0000-0001-8753-466X