Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats.
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Stress-Induced Reinstatement of Drug Seeking: 20 Years of Progress.The effects of pioglitazone, a PPARγ receptor agonist, on the abuse liability of oxycodone among nondependent opioid users.PPAR agonists regulate brain gene expression: relationship to their effects on ethanol consumptionNeuroimmune pathways in alcohol consumption: evidence from behavioral and genetic studies in rodents and humansPeroxisome proliferator-activated receptors α and γ are linked with alcohol consumption in mice and withdrawal and dependence in humansPPARγ activation attenuates opioid consumption and modulates mesolimbic dopamine transmissionFetal alcohol spectrum disorders and neuroimmune changesPPAR Agonists: I. Role of Receptor Subunits in Alcohol Consumption in Male and Female Mice.Preclinical Medication Development: New Targets and New Drugs.Peroxisome proliferator-activated receptor (PPAR) agonists as promising new medications for drug addiction: preclinical evidence.Emerging targets for addiction neuropharmacology: From mechanisms to therapeuticsCombined pharmacotherapies for the management of alcoholism: rationale and evidence to date.Investigational therapies for the pharmacological treatment of alcoholism.Inflammatory responses to alcohol in the CNS: nuclear receptors as potential therapeutics for alcohol-induced neuropathologiesGenetic Deletion of Neuronal PPARγ Enhances the Emotional Response to Acute Stress and Exacerbates Anxiety: An Effect Reversed by Rescue of Amygdala PPARγ Function.Localization of PPAR isotypes in the adult mouse and human brain.Glial and neuroinflammatory targets for treating substance use disorders.Analgesic tolerance to morphine is regulated by PPARγ.PPAR-gamma agonist pioglitazone modifies craving intensity and brain white matter integrity in patients with primary cocaine use disorder: a double-blind randomized controlled pilot trial.Multi-modal MRI classifiers identify excessive alcohol consumption and treatment effects in the brain.Ethanol and Cytokines in the Central Nervous System.Inhibition of fatty acid amide hydrolase in the central amygdala alleviates co-morbid expression of innate anxiety and excessive alcohol intake.Protection against alcohol-induced neuronal and cognitive damage by the PPARγ receptor agonist pioglitazone.PPARγ agonism attenuates cocaine cue reactivity.Pioglitazone, a PPARγ agonist, reduces nicotine craving in humans, with marginal effects on abuse potential.Peroxisome Proliferator Activated Receptor Agonists Modulate Transposable Element Expression in Brain and LiverAnti-stress neuropharmacological mechanisms and targets for addiction treatment: A translational framework
P2860
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P2860
Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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2013年學術文章
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name
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@en
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@nl
type
label
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@en
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@nl
prefLabel
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@en
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@nl
P2093
P2860
P50
P356
P1476
Activation of PPARγ by pioglit ...... nking and relapse in msP rats.
@en
P2093
Andrea Cippitelli
George Gaitanaris
Gregory Demopulos
Lorenzo Somaini
Roberto Ciccocioppo
Serena Stopponi
P2860
P304
P356
10.1111/ACER.12091
P577
2013-03-29T00:00:00Z