Endoplasmic reticulum stress contributes to heart protection induced by cyclophilin D inhibition.
about
The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotectionATF6 mediates a pro-inflammatory synergy between ER stress and TLR activation in the pathogenesis of liver ischemia-reperfusion injury.bFGF attenuates endoplasmic reticulum stress and mitochondrial injury on myocardial ischaemia/reperfusion via activation of PI3K/Akt/ERK1/2 pathway.Tauroursodeoxycholic acid inhibits endoplasmic reticulum stress, blocks mitochondrial permeability transition pore opening, and suppresses reperfusion injury through GSK-3ß in cardiac H9c2 cells.Peptidyl-prolyl isomerases: a full cast of critical actors in cardiovascular diseases.Mitochondrial Cyclophilin D as a Potential Therapeutic Target for Ischemia-Induced Facial Palsy in Rats.
P2860
Endoplasmic reticulum stress contributes to heart protection induced by cyclophilin D inhibition.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@en
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@nl
type
label
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@en
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@nl
prefLabel
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@en
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@nl
P2093
P2860
P1476
Endoplasmic reticulum stress c ...... d by cyclophilin D inhibition.
@en
P2093
Annie Durand
Elise Belaidi
Johanna Decorps
Lionel Augeul
Michel Ovize
P2860
P2888
P356
10.1007/S00395-013-0363-Z
P577
2013-06-07T00:00:00Z