Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals.
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Alterations in Mitochondrial Quality Control in Alzheimer's DiseaseMitochondria in motor nerve terminals: function in health and in mutant superoxide dismutase 1 mouse models of familial ALSMitochondrial transport in neurons: impact on synaptic homeostasis and neurodegeneration.Organelle-Specific Sensors for Monitoring Ca2+ Dynamics in NeuronsDevelopmental mechanisms for suppressing the effects of delayed release at the endbulb of Held.Spatiotemporal regulation of ATP and Ca2+ dynamics in vertebrate rod and cone ribbon synapses.Mitochondrial Aspects of Synaptic Dysfunction in Alzheimer's Disease.Activation of protein kinase C delta following cerebral ischemia leads to release of cytochrome C from the mitochondria via bad pathway.Synaptic vesicle exocytosis in hippocampal synaptosomes correlates directly with total mitochondrial volume.Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease.The mitochondrial complex V-associated large-conductance inner membrane current is regulated by cyclosporine and dexpramipexoleRepetitive nerve stimulation transiently opens the mitochondrial permeability transition pore in motor nerve terminals of symptomatic mutant SOD1 miceDeletion of a kinesin I motor unmasks a mechanism of homeostatic branching control by neurotrophin-3.Genetic analysis in Drosophila reveals a role for the mitochondrial protein p32 in synaptic transmissionPhysiology and pathology of calcium signaling in the brain.Synaptic mitochondrial pathology in Alzheimer's diseaseLKB1 Regulates Mitochondria-Dependent Presynaptic Calcium Clearance and Neurotransmitter Release Properties at Excitatory Synapses along Cortical AxonsHomocysteine aggravates ROS-induced depression of transmitter release from motor nerve terminals: potential mechanism of peripheral impairment in motor neuron diseases associated with hyperhomocysteinemia.Mitochondria and neurotransmission: evacuating the synapse.Morphological and functional changes in innervation of a fast forelimb muscle in SOD1-G85R mice.Increased asynchronous release and aberrant calcium channel activation in amyloid precursor protein deficient neuromuscular synapses.Mitochondria-Targeted Antioxidant SS31 Prevents Amyloid Beta-Induced Mitochondrial Abnormalities and Synaptic Degeneration in Alzheimer's DiseaseCytosolic calcium coordinates mitochondrial energy metabolism with presynaptic activityMitochondrial Calcium Uptake Modulates Synaptic Vesicle Endocytosis in Central Nerve TerminalsMitochondria at the synapse.Synaptic transmission at parasympathetic neurons of the major pelvic ganglion from normal and diabetic male mice.Frequency-dependent modes of synaptic vesicle endocytosis and exocytosis at adult mouse neuromuscular junctionsMitochondrial calcium uptake.The Psi(m) depolarization that accompanies mitochondrial Ca2+ uptake is greater in mutant SOD1 than in wild-type mouse motor terminals.Expression of human uncoupling protein-3 in Drosophila insulin-producing cells increases insulin-like peptide (DILP) levels and shortens lifespan.Transmitochondrial embryonic stem cells containing pathogenic mtDNA mutations are compromised in neuronal differentiation.Mitochondrial calcium function and dysfunction in the central nervous system.Mitochondrial trafficking and anchoring in neurons: New insight and implications.Contributions of Bcl-xL to acute and long term changes in bioenergetics during neuronal plasticity.Molecular mechanisms for synchronous, asynchronous, and spontaneous neurotransmitter release.Bcl-xL in neuroprotection and plasticity.Mitochondrial Dysfunction and Synaptic Transmission Failure in Alzheimer's Disease.The Interplay of Axonal Energy Homeostasis and Mitochondrial Trafficking and Anchoring.Asynchronous presynaptic glutamate release enhances neuronal excitability during the post-spike refractory period.Mitochondrial Ca(2+) Processing by a Unit of Mitochondrial Ca(2+) Uniporter and Na(+)/Ca(2+) Exchanger Supports the Neuronal Ca(2+) Influx via Activated Glutamate Receptors.
P2860
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P2860
Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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name
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@en
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@nl
type
label
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@en
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@nl
prefLabel
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@en
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@nl
P2860
P1476
Mitochondrial Ca2+ uptake prev ...... f mouse motor nerve terminals.
@en
P2093
Ellen F Barrett
Gavriel David
P2860
P304
P356
10.1113/JPHYSIOL.2002.035196
P407
P577
2003-02-14T00:00:00Z