Targeted gene replacement of a ferredoxin gene in Trichomonas vaginalis does not lead to metronidazole resistance.
about
Draft genome sequence of the sexually transmitted pathogen Trichomonas vaginalisTrichomoniasisTreatment of infections caused by metronidazole-resistant Trichomonas vaginalisExtensive genetic diversity, unique population structure and evidence of genetic exchange in the sexually transmitted parasite Trichomonas vaginalisBiochemistry and evolution of anaerobic energy metabolism in eukaryotesStructural basis of 5-nitroimidazole antibiotic resistance: the crystal structure of NimA from Deinococcus radiodurans.Current therapeutics, their problems, and sulfur-containing-amino-acid metabolism as a novel target against infections by "amitochondriate" protozoan parasites.The Trichomonas vaginalis hydrogenosome proteome is highly reduced relative to mitochondria, yet complex compared with mitosomesMolecular dynamics simulations of Trichomonas vaginalis ferredoxin show a loop-cap transitionNovel insights into the molecular events linking to cell death induced by tetracycline in the amitochondriate protozoan Trichomonas vaginalis.Silencing the ap65 gene reduces adherence to vaginal epithelial cells by Trichomonas vaginalis.Hydrogenosome metabolism is the key target for antiparasitic activity of resveratrol against Trichomonas vaginalis.Ferredoxin Gene Mutation in Iranian Trichomonas vaginalis IsolatesTrichomonas vaginalis: An Updated Overview Towards Diagnostic Improvement.The actin-based machinery of Trichomonas vaginalis mediates flagellate-amoeboid transition and migration across host tissue.Genetic Indicators of Drug Resistance in the Highly Repetitive Genome of Trichomonas vaginalis.Alternative pathway of metronidazole activation in Trichomonas vaginalis hydrogenosomes.Eicosapentaenoic Acid Modulates Trichomonas vaginalis Activity.Protein import into hydrogenosomes of Trichomonas vaginalis involves both N-terminal and internal targeting signals: a case study of thioredoxin reductases.Conservation of Transit Peptide-Independent Protein Import into the Mitochondrial and Hydrogenosomal Matrix.Knockout of the abundant Trichomonas vaginalis hydrogenosomal membrane protein TvHMP23 increases hydrogenosome size but induces no compensatory up-regulation of paralogous copies.CRISPR/Cas9-mediated gene modification and gene knock out in the human-infective parasite Trichomonas vaginalis.Use of Trichomonas vaginalis clinical isolates to evaluate correlation of gene expression and metronidazole resistance.The CRISPR/Cas9 system sheds new lights on the biology of protozoan parasites.The N-Terminal Sequences of Four Major Hydrogenosomal Proteins Are Not Essential for Import into Hydrogenosomes ofTrichomonas vaginalis
P2860
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P2860
Targeted gene replacement of a ferredoxin gene in Trichomonas vaginalis does not lead to metronidazole resistance.
description
2004 nî lūn-bûn
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name
Targeted gene replacement of a ...... d to metronidazole resistance.
@en
Targeted gene replacement of a ...... d to metronidazole resistance.
@nl
type
label
Targeted gene replacement of a ...... d to metronidazole resistance.
@en
Targeted gene replacement of a ...... d to metronidazole resistance.
@nl
prefLabel
Targeted gene replacement of a ...... d to metronidazole resistance.
@en
Targeted gene replacement of a ...... d to metronidazole resistance.
@nl
P2093
P50
P1476
Targeted gene replacement of a ...... d to metronidazole resistance.
@en
P2093
Christine L Hsieh
Jan Tachezy
Kirkwood M Land
Maria G Delgadillo-Correa
P2860
P304
P356
10.1046/J.1365-2958.2003.03791.X
P407
P577
2004-01-01T00:00:00Z