Cutting edge: Inhibition of IL-6 trans-signaling protects from malaria-induced lethality in mice.
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Mechanisms of chronic JAK-STAT3-SOCS3 signaling in obesitySignaling by IL-6 promotes alternative activation of macrophages to limit endotoxemia and obesity-associated resistance to insulin.Liver-inherent immune system: its role in blood-stage malaria.BID-dependent release of mitochondrial SMAC dampens XIAP-mediated immunity against Shigella.Reductions in microfilaridermia by repeated ivermectin treatment are associated with lower Plasmodium-specific Th17 immune responses in Onchocerca volvulus-infected individuals.Effect of mushroom Agaricus blazei on immune response and development of experimental cerebral malaria.Promotion of Expansion and Differentiation of Hematopoietic Stem Cells by Interleukin-27 into Myeloid Progenitors to Control Infection in Emergency Myelopoiesis.T cell subtypes and reciprocal inflammatory mediator expression differentiate P. falciparum memory recall responses in asymptomatic and symptomatic malaria patients in southeastern Haiti.Protective Vaccination against Blood-Stage Malaria of Plasmodium chabaudi: Differential Gene Expression in the Liver of Balb/c Mice toward the End of Crisis Phase.Towards identifying novel anti-Eimeria agents: trace elements, vitamins, and plant-based natural products.Protective vaccination and blood-stage malaria modify DNA methylation of gene promoters in the liver of Balb/c mice.IL-6 promotes CD4+ T-cell and B-cell activation during Plasmodium infection.Genome-wide screening identifies Plasmodium chabaudi-induced modifications of DNA methylation status of Tlr1 and Tlr6 gene promoters in liver, but not spleen, of female C57BL/6 mice.Obesity exacerbates colitis-associated cancer via IL-6-regulated macrophage polarisation and CCL-20/CCR-6-mediated lymphocyte recruitment.Elevated plasma abscisic acid is associated with asymptomatic falciparum malaria and with IgG-/caspase-1-dependent immunity in Plasmodium yoelii-infected mice.Gene expression of the liver of vaccination-protected mice in response to early patent infections of Plasmodium chabaudi blood-stage malaria.Hepatic leptin receptor expression can partially compensate for IL-6Rα deficiency in DEN-induced hepatocellular carcinoma
P2860
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P2860
Cutting edge: Inhibition of IL-6 trans-signaling protects from malaria-induced lethality in mice.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@en
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@nl
type
label
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@en
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@nl
prefLabel
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@en
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@nl
P2093
P50
P356
P1476
Cutting edge: Inhibition of IL ...... ria-induced lethality in mice.
@en
P2093
Bhagirath Chaurasia
Claudia M Wunderlich
F Thomas Wunderlich
Kristina Behnke
Peter Ströhle
P304
P356
10.4049/JIMMUNOL.1102137
P407
P577
2012-03-30T00:00:00Z