Conducted dilations initiated by purines in arterioles are endothelium dependent and require endothelial Ca2+.
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The vascular conducted response in cerebral blood flow regulationContribution of non-endothelium-dependent substances to exercise hyperaemia: are they O(2) dependent?Hyperinsulinemia: effect on cardiac mass/function, angiotensin II receptor expression, and insulin signaling pathwaysAdenosine A2A receptor modulation of juvenile female rat skeletal muscle microvessel permeabilityPannexin channel and connexin hemichannel expression in vascular function and inflammationEnhanced spontaneous Ca2+ events in endothelial cells reflect signalling through myoendothelial gap junctions in pressurized mesenteric arteriesKIR channel activation contributes to onset and steady-state exercise hyperemia in humans.Adenine nucleotide control of coronary blood flow during exerciseCapillary endothelial cells as coordinators of skeletal muscle blood flow during active hyperemia.A critical role for the vascular endothelium in functional neurovascular coupling in the brain.Control of muscle blood flow during exercise: local factors and integrative mechanismsVisualizing calcium responses to acetylcholine convection along endothelium of arteriolar networks in Cx40BAC-GCaMP2 transgenic miceMechanisms of ATP-mediated vasodilation in humans: modest role for nitric oxide and vasodilating prostaglandins.Purinergic glio-endothelial coupling during neuronal activity: role of P2Y1 receptors and eNOS in functional hyperemia in the mouse somatosensory cortex.Theoretical model of metabolic blood flow regulation: roles of ATP release by red blood cells and conducted responsesEffect of extraluminal ATP application on vascular tone and blood flow in skeletal muscle: implications for exercise hyperemia.What is the efficiency of ATP signaling from erythrocytes to regulate distribution of O(2) supply within the microvasculature?Mechanism of ATP-induced local and conducted vasomotor responses in isolated rat cerebral penetrating arterioles.Contribution of intravascular versus interstitial purines and nitric oxide in the regulation of exercise hyperaemia in humans.Vasodilator interactions in skeletal muscle blood flow regulation.Flow-induced ATP release in patient-specific arterial geometries--a comparative study of computational models.Regulation of the skeletal muscle blood flow in humans.Augmentation of Muscle Blood Flow by Ultrasound Cavitation Is Mediated by ATP and Purinergic Signaling.Local control of blood flow during active hyperaemia: what kinds of integration are important?Vascular function in health, hypertension, and diabetes: effect of physical activity on skeletal muscle microcirculation.Cardiovascular Adaptations to Exercise Training.Endothelial-smooth muscle cell interactions in the regulation of vascular tone in skeletal muscle.Cyclic nucleotide-gated channels: a familiar channel family with a new function?Conducted dilatation to ATP and K+ in rat skeletal muscle arteriolesFrom one generation to the next: a comprehensive account of sympathetic receptor control in branching arteriolar trees.Localized transient increases in endothelial cell Ca2+ in arterioles in situ: implications for coordination of vascular function.P2Y receptors as regulators of lung endothelial barrier integrity.Different pathways with distinct properties conduct dilations in the microcirculation in vivo.Increase in endothelial cell Ca(2+) in response to mouse cremaster muscle contraction.Plasma ATP during exercise: possible role in regulation of coronary blood flow.ATP-induced vasodilation and purinergic receptors in the human leg: roles of nitric oxide, prostaglandins, and adenosine.Integrative model of coronary flow in anatomically based vasculature under myogenic, shear, and metabolic regulation.Sympatholytic effect of intravascular ATP is independent of nitric oxide, prostaglandins, Na+ /K+ -ATPase and KIR channels in humans.Role of myoendothelial communication on arterial vasomotion.Mechanisms initiating integrin-stimulated flow recruitment in arteriolar networks.
P2860
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P2860
Conducted dilations initiated by purines in arterioles are endothelium dependent and require endothelial Ca2+.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Conducted dilations initiated ...... and require endothelial Ca2+.
@en
Conducted dilations initiated ...... and require endothelial Ca2+.
@nl
type
label
Conducted dilations initiated ...... and require endothelial Ca2+.
@en
Conducted dilations initiated ...... and require endothelial Ca2+.
@nl
prefLabel
Conducted dilations initiated ...... and require endothelial Ca2+.
@en
Conducted dilations initiated ...... and require endothelial Ca2+.
@nl
P2860
P1476
Conducted dilations initiated ...... and require endothelial Ca2+.
@en
P2093
Ingrid H Sarelius
Tasmia Duza
P2860
P304
P356
10.1152/AJPHEART.00788.2002
P577
2003-03-13T00:00:00Z