Overexpression of human chorionic gonadotropin causes multiple reproductive defects in transgenic mice.
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Genetic Models for the Study of Luteinizing Hormone Receptor FunctionInfertility in Female Mice with a Gain-of-Function Mutation in the Luteinizing Hormone Receptor Is Due to Irregular Estrous Cyclicity, Anovulation, Hormonal Alterations, and Polycystic Ovaries.Redirecting intracellular trafficking and the secretion pattern of FSH dramatically enhances ovarian function in mice.Precocious puberty and Leydig cell hyperplasia in male mice with a gain of function mutation in the LH receptor gene.Ablation of GalNAc-4-sulfotransferase-1 enhances reproduction by altering the carbohydrate structures of luteinizing hormone in miceTargeted disruption of luteinizing hormone beta-subunit leads to hypogonadism, defects in gonadal steroidogenesis, and infertilityTransgenic mouse technology: principles and methodshCG-induced endoplasmic reticulum stress triggers apoptosis and reduces steroidogenic enzyme expression through activating transcription factor 6 in Leydig cells of the testisPituitary-specific overexpression of porcine follicle-stimulating hormone leads to improvement of female fecundity in BAC transgenic mice.Luteinizing hormone receptor deficiency increases the susceptibility to alkylating agent-induced lymphomagenesis in mice.Animal models for aberrations of gonadotropin action.Phthalate-induced Leydig cell hyperplasia is associated with multiple endocrine disturbances.Constitutively active luteinizing hormone receptors: consequences of in vivo expression.The intraovarian actions of estrogen receptor-alpha are necessary to repress the formation of morphological and functional Leydig-like cells in the female gonad.Gonadectomy-induced adrenocortical neoplasia in the domestic ferret (Mustela putorius furo) and laboratory mouse.Genetic modeling of ovarian phenotypes in mice for the study of human polycystic ovary syndromeFollistatin-like 3 (FSTL3) mediated silencing of transforming growth factor β (TGFβ) signaling is essential for testicular aging and regulating testis sizePTTG: an important target gene for ovarian cancer therapy.A unique human chorionic gonadotropin antagonist suppresses ovarian hyperstimulation syndrome in rats.The mammalian ovary from genesis to revelation.Luteinizing hormone and human chorionic gonadotropin: distinguishing unique physiologic rolesThe role of Notch signaling in the mammalian ovary.Human chorionic gonadotropin induces human macrophages to form intracytoplasmic vacuoles mimicking Hofbauer cells in human chorionic villi.Luteinizing hormone receptor-mediated effects on initiation of spermatogenesis in gonadotropin-deficient (hpg) mice are replicated by testosterone.P-glycoprotein and breast cancer resistance protein expression in human placentae of various gestational ages.Fetal but not adult Leydig cells are susceptible to adenoma formation in response to persistently high hCG level: a study on hCG overexpressing transgenic mice.Constitutive luteinizing hormone receptor signaling causes sexual dysfunction and Leydig cell adenomas in male mice.
P2860
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P2860
Overexpression of human chorionic gonadotropin causes multiple reproductive defects in transgenic mice.
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2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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name
Overexpression of human chorio ...... ve defects in transgenic mice.
@en
Overexpression of human chorio ...... ve defects in transgenic mice.
@nl
type
label
Overexpression of human chorio ...... ve defects in transgenic mice.
@en
Overexpression of human chorio ...... ve defects in transgenic mice.
@nl
prefLabel
Overexpression of human chorio ...... ve defects in transgenic mice.
@en
Overexpression of human chorio ...... ve defects in transgenic mice.
@nl
P2093
P2860
P1476
Overexpression of human chorio ...... ive defects in transgenic mice
@en
P2093
Lou Ann Hadsell
Martin M Matzuk
T Rajendra Kumar
P2860
P304
P356
10.1095/BIOLREPROD.102.013953
P407
P577
2003-04-02T00:00:00Z