Induction of T-type calcium channel gene expression by chronic hypoxia.
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Ca2+-activated K+ channels in human melanoma cells are up-regulated by hypoxia involving hypoxia-inducible factor-1alpha and the von Hippel-Lindau proteinFunctional and transcriptional induction of aquaporin-1 gene by hypoxia; analysis of promoter and role of Hif-1αT-type calcium channels blockers as new tools in cancer therapiesOxygen tension regulates mitochondrial DNA-encoded complex I gene expressionIL1-receptor accessory protein-like 1 (IL1RAPL1), a protein involved in cognitive functions, regulates N-type Ca2+-channel and neurite elongationDown-regulation of interleukin 7 mRNA by hypoxia is calcium dependentInhibition of T-type calcium current in the reticular thalamic nucleus by a novel neuroactive steroid.Allethrin differentially modulates voltage-gated calcium channel subtypes in rat PC12 cells.Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cells.Transcriptional regulation of α1H T-type calcium channel under hypoxiaHypoxia. 4. Hypoxia and ion channel functionNNC 55-0396, a T-type Ca2+ channel inhibitor, inhibits angiogenesis via suppression of hypoxia-inducible factor-1α signal transduction.Hypoxia-inducible factor-1α regulates the expression of L-type voltage-dependent Ca(2+) channels in PC12 cells under hypoxia.Regulation of oxygen sensing by ion channels.Developmental and stress-induced remodeling of cell–cell communication in the adrenal medullary tissue.Mitochondrial Ca2+ uptake from plasma membrane Cav3.2 protein channels contributes to ischemic toxicity in PC12 cellsFunctional chromaffin cell plasticity in response to stress: focus on nicotinic, gap junction, and voltage-gated Ca2+ channels.Upregulation of vascular calcium channels in neonatal piglets with hypoxia-induced pulmonary hypertension.T-type calcium channels are regulated by hypoxia/reoxygenation in ventricular myocytes.T-type Ca2+ channels promote oxygenation-induced closure of the rat ductus arteriosus not only by vasoconstriction but also by neointima formation.Adaptive and maladaptive cardiorespiratory responses to continuous and intermittent hypoxia mediated by hypoxia-inducible factors 1 and 2.Peripheral chemoreceptors: function and plasticity of the carotid body.T-type channel-mediated neurotransmitter release.Gene expression analyses reveal metabolic specifications in acute O2 -sensing chemoreceptor cells.Oxygen sensing by the carotid body: mechanisms and role in adaptation to hypoxia.Enhanced BDNF signalling following chronic hypoxia potentiates catecholamine release from cultured rat adrenal chromaffin cells.In vitro study of normoxic epidermal growth factor receptor-induced hypoxia-inducible factor-1-alpha, vascular endothelial growth factor, and BNIP3 expression in head and neck squamous cell carcinoma cell lines: Implications for anti-epidermal growtTranscriptional regulation of voltage-gated Ca2+ channels.Functional expression of voltage-gated calcium channels in human melanoma.T-type Ca2+ channels in mouse embryonic stem cells: modulation during cell cycle and contribution to self-renewal.Involvement of focal adhesion kinase in cellular invasion of head and neck squamous cell carcinomas via regulation of MMP-2 expression.Epstein-Barr virus transformation of human lymphoblastoid cells from patients with fragile X syndrome induces variable changes on CGG repeats size and promoter methylation.The effect of oxygen tension on calcium homeostasis in bovine articular chondrocytes.The role of ion channels in the hypoxia-induced aggressiveness of glioblastoma.Molecular mechanisms of Ca(2+) signaling in neurons induced by the S100A4 protein.Gene Expressions Underlying Mishandled Calcium Clearance and Elevated Generation of Reactive Oxygen Species in the Coronary Artery Smooth Muscle Cells of Chronic Heart Failure Rats.Hexabromocyclododecane inhibits depolarization-induced increase in intracellular calcium levels and neurotransmitter release in PC12 cells.Carotid body chemosensory responses in mice deficient of TASK channels.Calcium signaling and T-type calcium channels in cancer cell cycling.Magnesium deficiency causes loss of response to intermittent hypoxia in paraganglion cells.
P2860
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P2860
Induction of T-type calcium channel gene expression by chronic hypoxia.
description
2003 nî lūn-bûn
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2003年の論文
@ja
2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
@zh-hant
name
Induction of T-type calcium channel gene expression by chronic hypoxia.
@en
Induction of T-type calcium channel gene expression by chronic hypoxia.
@nl
type
label
Induction of T-type calcium channel gene expression by chronic hypoxia.
@en
Induction of T-type calcium channel gene expression by chronic hypoxia.
@nl
prefLabel
Induction of T-type calcium channel gene expression by chronic hypoxia.
@en
Induction of T-type calcium channel gene expression by chronic hypoxia.
@nl
P2093
P2860
P356
P1476
Induction of T-type calcium channel gene expression by chronic hypoxia.
@en
P2093
José López-Barneo
Konstantin L Levitsky
María D Chiara
Raquel Del Toro
P2860
P304
22316-22324
P356
10.1074/JBC.M212576200
P407
P577
2003-04-04T00:00:00Z