Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
about
The spontaneous appearance rate of the yeast prion [PSI+] and its implications for the evolution of the evolvability properties of the [PSI+] systemProbing the role of structural features of mouse PrP in yeast by expression as Sup35-PrP fusionsHsp104-dependent remodeling of prion complexes mediates protein-only inheritanceRole of a ribosome-associated E3 ubiquitin ligase in protein quality controlTranslation initiation factors eIF3 and HCR1 control translation termination and stop codon read-through in yeast cellsHeterologous cross-seeding mimics cross-species prion conversion in a yeast model.Inactivation of NMD increases viability of sup45 nonsense mutants in Saccharomyces cerevisiae.Prion formation and polyglutamine aggregation are controlled by two classes of genes.Development and validation of a yeast high-throughput screen for inhibitors of Aβ₄₂ oligomerization.Genetic basis of hidden phenotypic variation revealed by increased translational readthrough in yeast.J-protein co-chaperone Sis1 required for generation of [RNQ+] seeds necessary for prion propagationThe Paf1 complex subunit Rtf1 buffers cells against the toxic effects of [PSI+] and defects in Rkr1-dependent protein quality control in Saccharomyces cerevisiae.[PSI(+) ] prion variant establishment in yeast"Prion-proof" for [PIN+]: infection with in vitro-made amyloid aggregates of Rnq1p-(132-405) induces [PIN+]An engineered nonsense URA3 allele provides a versatile system to detect the presence, absence and appearance of the [PSI+] prion in Saccharomyces cerevisiaeExploring the basis of [PIN(+)] variant differences in [PSI(+)] inductionMutants of the Paf1 complex alter phenotypic expression of the yeast prion [PSI+]Antiprion drugs as chemical tools to uncover mechanisms of prion propagation.Ubiquitylation by the Ltn1 E3 ligase protects 60S ribosomes from starvation-induced selective autophagyInhibition of Aβ42 oligomerization in yeast by a PICALM ortholog and certain FDA approved drugs.Prion-dependent lethality of sup45 mutants in Saccharomyces cerevisiae.Epigenetic regulation of translation reveals hidden genetic variation to produce complex traits
P2860
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P2860
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
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2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
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2003年學術文章
@zh-hant
name
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@en
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@nl
type
label
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@en
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@nl
prefLabel
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@en
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@nl
P2093
P356
P1433
P1476
Guanidine reduces stop codon read-through caused by missense mutations in SUP35 or SUP45.
@en
P2093
Michael E Bradley
Namitha Vishveshwara
Sviatoslav Bagriantsev
P2860
P304
P356
10.1002/YEA.985
P577
2003-05-01T00:00:00Z