Excessive fuel availability amplifies the FTO-mediated obesity risk: results from the TUEF and Whitehall II studies.

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Excessive fuel availability amplifies the FTO-mediated obesity risk: results from the TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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Excessive fuel availability am ...... TUEF and Whitehall II studies.

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P1476

Excessive fuel availability am ...... TUEF and Whitehall II studies

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Andreas Fritsche

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Andreas Peter

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Benjamin A Jaghutriz

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Ellen Fehlert

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Fausto Machicao

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Hans-Ulrich Häring

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Louise Fritsche

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Róbert J Bánhegyi

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P2860

Genetic studies of body mass index yield new insights for obesity biology

FTO Obesity Variant Circuitry and Adipocyte Browning in Humans

The bigger picture of FTO: the first GWAS-identified obesity gene

Gene-centric association signals for lipids and apolipoproteins identified via the HumanCVD BeadChip

Obesity-associated variants within FTO form long-range functional connections with IRX3

Variation in the FTO gene influences food intake but not energy expenditure

Variation in the obesity risk gene FTO determines the postprandial cerebral processing of food stimuli in the prefrontal cortex.

A simulation study of gene-by-environment interactions in GWAS implies ample hidden effects.

Physical activity attenuates the influence of FTO variants on obesity risk: a meta-analysis of 218,166 adults and 19,268 children.

Hepatic and whole-body fat synthesis in humans during carbohydrate overfeeding.

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s41598-017-15744-4

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scholarly article

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10.1038/S41598-017-15744-4

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English

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2017-11-14T00:00:00Z

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29138452

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Adipositas

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5686126

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