iNOS activity is essential for endothelial stress gene expression protecting against oxidative damage. - Wikidata - wikimedia - TriplyDB

iNOS activity is essential for endothelial stress gene expression protecting against oxidative damage.

iNOS activity is essential for endothelial stress gene expression protecting against oxidative damage.

http://www.wikidata.org/entity/Q44527143

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Impact of Superoxide Dismutase Mimetic AEOL 10150 on the Endothelin System of Fischer 344 Rats A heptameric peptide purified from Spirulina sp. gastrointestinal hydrolysate inhibits angiotensin I-converting enzyme- and angiotensin II-induced vascular dysfunction in human endothelial cells Mitohormesis: Promoting Health and Lifespan by Increased Levels of Reactive Oxygen Species (ROS).Suppression of Transforming Growth Factor-β Signaling Delays Cellular Senescence and Preserves the Function of Endothelial Cells Derived from Human Pluripotent Stem Cells.N-Acetylcysteine increases corneal endothelial cell survival in a mouse model of Fuchs endothelial corneal dystrophy Racial differences in oxidative stress and inflammation: in vitro and in vivo.Pharmacological modulation of dietary lipid-induced cerebral capillary dysfunction: Considerations for reducing risk for Alzheimer's disease.Endothelial glutathione-S-transferase A4-4 protects against oxidative stress and modulates iNOS expression through NF-kappaB translocation.Evaluation of a synthetic CArG promoter for nitric oxide synthase gene therapy of cancer.Nitric oxide modulates vascular inflammation and intimal hyperplasia in insulin resistance and the metabolic syndrome.

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iNOS activity is essential for endothelial stress gene expression protecting against oxidative damage.

http://www.wikidata.org/entity/Q44527143

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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iNOS activity is essential for ...... ting against oxidative damage.

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iNOS activity is essential for ...... ting against oxidative damage.

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iNOS activity is essential for ...... ting against oxidative damage.

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iNOS activity is essential for ...... ting against oxidative damage.

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iNOS activity is essential for ...... ting against oxidative damage.

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JAPPLPHYSIOL.00419.2003

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Journal of Applied Physiology

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iNOS activity is essential for ...... ting against oxidative damage.

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Christoph V Suschek

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Guido Lerzynski

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Karsten Hemmrich

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Victoria Kolb-Bachofen

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P2860

VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodia

Inducible nitric oxide synthase and vascular injury.

Nitric oxide can inhibit apoptosis or switch it into necrosis.

Implications of inducible nitric oxide synthase expression and enzyme activity.

Vascular effects of dietary L-arginine supplementation.

Reprogramming of the macrophage transcriptome in response to interferon-gamma and Mycobacterium tuberculosis: signaling roles of nitric oxide synthase-2 and phagocyte oxidase

Inducible nitric oxide synthase suppresses the development of allograft arteriosclerosis.

Nitric oxide decreases the sensitivity of pulmonary endothelial cells to LPS-induced apoptosis in a zinc-dependent fashion.

The effect of antisense oligodeoxynucleotides on nitric oxide secretion from macrophage-like cells.

Nitric oxide mediates intracytoplasmic and intranuclear zinc release.

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