PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor. - Wikidata - wikimedia - TriplyDB

PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor.

PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor.

http://www.wikidata.org/entity/Q44535674

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Characteristics of a large cohort of patients with autoimmune pulmonary alveolar proteinosis in Japan.Alcohol Use As a Risk Factor in Infections and Healing: A Clinician's Perspective GM-CSF receptor expression and signaling is decreased in lungs of ethanol-fed rats Why does the autoantibody against granulocyte-macrophage colony-stimulating factor cause lesions only in the lung?Granulocyte-macrophage colony stimulating factor-mediated innate responses in tuberculosis Downregulation of PU.1 leads to decreased expression of Dectin-1 in alveolar macrophages during Pneumocystis pneumonia.The molecular basis of pulmonary alveolar proteinosis Patient-derived granulocyte/macrophage colony-stimulating factor autoantibodies reproduce pulmonary alveolar proteinosis in nonhuman primates.Delayed neonatal lung macrophage differentiation in a mouse model of in utero ethanol exposure.In vitro effector activity of Pneumocystis murina-specific T-cytotoxic-1 CD8+ T cells: role of granulocyte-macrophage colony-stimulating factor.Interleukin-3 receptor in acute leukemia.Adherent human alveolar macrophages exhibit a transient pro-inflammatory profile that confounds responses to innate immune stimulation Hereditary pulmonary alveolar proteinosis: pathogenesis, presentation, diagnosis, and therapy.Pulmonary macrophage transplantation therapy Immunodeficiency secondary to anticytokine autoantibodies Functional PU.1 in macrophages has a pivotal role in NF-κB activation and neutrophilic lung inflammation during endotoxemia.PU.1 is involved in the immune response to Aspergillus fumigatus through upregulating Dectin-1 expression.Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.Alveolar macrophage cathelicidin deficiency in severe sarcoidosis.Absence of the cystic fibrosis transmembrane regulator (Cftr) from myeloid-derived cells slows resolution of inflammation and infection.Integrin beta6 mediates phospholipid and collectin homeostasis by activation of latent TGF-beta1.HIV-1-transgene expression in rats decreases alveolar macrophage zinc levels and phagocytosis.Alveolar Epithelial Cell-Derived Prostaglandin E2 Serves as a Request Signal for Macrophage Secretion of Suppressor of Cytokine Signaling 3 during Innate Inflammation.Out of breath: GM-CSFRalpha mutations disrupt surfactant homeostasis.Autoantibody-Mediated Pulmonary Alveolar Proteinosis in Rasgrp1-Deficient Mice.Macrophage differentiation and function in health and disease.Airway inflammatory cell responses to intra-amniotic lipopolysaccharide in a sheep model of chorioamnionitis Rare lung disease II: pulmonary alveolar proteinosis.Granulocyte-macrophage-colony-stimulating factor-dependent peritoneal macrophage responses determine survival in experimentally induced peritonitis and sepsis in mice.Is Cryptococcus gattii a Primary Pathogen?Pulmonary alveolar proteinosis, a primary immunodeficiency of impaired GM-CSF stimulation of macrophages.Use of induced pluripotent stem cells to recapitulate pulmonary alveolar proteinosis pathogenesis.Autoimmune pulmonary alveolar proteinosis: treatment options in year 2013.PU.1 is a major transcriptional activator of the tumour suppressor gene LIMD1.Gene expression analysis of myeloid and lymphoid lineage markers during mouse haematopoiesis.GM-CSF regulates a PU.1-dependent transcriptional program determining the pulmonary response to LPS.In Lysinuric Protein Intolerance system y+L activity is defective in monocytes and in GM-CSF-differentiated macrophages.The relative balance of GM-CSF and TGF-β1 regulates lung epithelial barrier function Targeted PPAR{gamma} deficiency in alveolar macrophages disrupts surfactant catabolism Zinc supplementation restores PU.1 and Nrf2 nuclear binding in alveolar macrophages and improves redox balance and bacterial clearance in the lungs of alcohol-fed rats.

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PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor.

http://www.wikidata.org/entity/Q44535674

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

@zh-hant

name

PU.1 regulation of human alveo ...... age colony-stimulating factor.

@en

PU.1 regulation of human alveo ...... age colony-stimulating factor.

@nl

type

label

PU.1 regulation of human alveo ...... age colony-stimulating factor.

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PU.1 regulation of human alveo ...... age colony-stimulating factor.

@nl

prefLabel

PU.1 regulation of human alveo ...... age colony-stimulating factor.

@en

PU.1 regulation of human alveo ...... age colony-stimulating factor.

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P356

AJPLUNG.00216.2003

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American Journal of Physiology - Lung Cellular and Molecular Physiology

P1476

PU.1 regulation of human alveo ...... age colony-stimulating factor.

@en

P2093

Anagha Malur

http://www.w3.org/2001/XMLSchema#string

Baisakhi Raychaudhuri

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Bruce C Trapnell

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Mani S Kavuru

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Mary Jane Thomassen

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Susamma Abraham

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Tracey L Bonfield

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P2860

Granulocyte/macrophage colony-stimulating factor-deficient mice show no major perturbation of hematopoiesis but develop a characteristic pulmonary pathology

Pulmonary alveolar proteinosis: progress in the first 44 years.

Gene therapy for human severe combined immunodeficiencies.

Gm-CSF regulates pulmonary surfactant homeostasis and alveolar macrophage-mediated innate host defense.

BAL findings in a patient with pulmonary alveolar proteinosis successfully treated with GM-CSF

Lungs of patients with idiopathic pulmonary alveolar proteinosis express a factor which neutralizes granulocyte-macrophage colony stimulating factor.

Idiopathic pulmonary alveolar proteinosis as an autoimmune disease with neutralizing antibody against granulocyte/macrophage colony-stimulating factor.

Nitric oxide inhibits inflammatory cytokine production by human alveolar macrophages.

Involvement of granulocyte-macrophage colony-stimulating factor in pulmonary homeostasis.

Exogenous granulocyte-macrophage colony-stimulating factor administration for pulmonary alveolar proteinosis.

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L1132-6

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10.1152/AJPLUNG.00216.2003

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5

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285

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2003-08-01T00:00:00Z

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10631060

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12896880

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P921