Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo. - Wikidata - wikimedia - TriplyDB

Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo.

Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo.

http://www.wikidata.org/entity/Q44540512

about

New and emerging disease modifying therapies for multiple sclerosis TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.Neuroprotective and anti-inflammatory effects of estrogen receptor ligand treatment in mice.Sodium MRI of multiple sclerosis.Emerging therapies in multiple sclerosis.Mitochondrial immobilization mediated by syntaphilin facilitates survival of demyelinated axons.Dynamics of sodium channel Nav1.5 expression in astrocytes in mouse models of multiple sclerosis Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger Oral administration of PF-01247324, a subtype-selective Nav1.8 blocker, reverses cerebellar deficits in a mouse model of multiple sclerosis Immunopathogenesis and immunotherapeutic approaches in multiple sclerosis.Inflammation, demyelination, neurodegeneration and neuroprotection in the pathogenesis of multiple sclerosis Sodium channels and multiple sclerosis: roles in symptom production, damage and therapy.Why do cannabinoids not show consistent effects as analgetic drugs in multiple sclerosis?Mechanisms of neuronal damage in multiple sclerosis and its animal models: role of calcium pumps and exchangers.Synthesis and evaluation of a 125I-labeled iminodihydroquinoline-derived tracer for imaging of voltage-gated sodium channels.Loss of Na+ channel beta2 subunits is neuroprotective in a mouse model of multiple sclerosis.Advancement of therapies for neuroprotection in multiple sclerosis.Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.An animal model of cortical and callosal pathology in multiple sclerosis.Approaches to neuroprotective strategies in multiple sclerosis.Mechanisms of neuronal dysfunction and degeneration in multiple sclerosis.Mitochondria as crucial players in demyelinated axons: lessons from neuropathology and experimental demyelination.Does long-term partial sodium channel blockade alter disease progression in MS? Evidence from a retrospective study.Mechanisms of neurodegeneration and axonal dysfunction in multiple sclerosis.Sodium channels in astroglia and microglia.Further understanding of the immunopathology of multiple sclerosis: impact on future treatments.Phenytoin: neuroprotection or neurotoxicity?Organotypic cultures of cerebellar slices as a model to investigate demyelinating disorders.Regulation of podosome formation in macrophages by a splice variant of the sodium channel SCN8A.Axonal protection achieved by blockade of sodium/calcium exchange in a new model of ischemia in vivo A recoverable state of axon injury persists for hours after spinal cord contusion in vivo.A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis.Nav1.5 sodium channels in macrophages in multiple sclerosis lesions.Experimental autoimmune encephalomyelitis from a tissue energy perspective.Safinamide and flecainide protect axons and reduce microglial activation in models of multiple sclerosis.Regulation and dysregulation of axon infrastructure by myelinating glia.Phenytoin: its potential as neuroprotective and retinoprotective drug.Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.Small nerve fibres, small hands and small feet: a new syndrome of pain, dysautonomia and acromesomelia in a kindred with a novel NaV1.7 mutation.Brain intra- and extracellular sodium concentration in multiple sclerosis: a 7 T MRI study.

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Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo.

http://www.wikidata.org/entity/Q44540512

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

Phenytoin protects spinal cord ...... of neuroinflammation in vivo.

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Phenytoin protects spinal cord ...... of neuroinflammation in vivo.

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Phenytoin protects spinal cord ...... of neuroinflammation in vivo.

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Phenytoin protects spinal cord ...... of neuroinflammation in vivo.

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Journal of Neurophysiology

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Phenytoin protects spinal cord ...... of neuroinflammation in vivo.

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Albert C Lo

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Carl Y Saab

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Joel A Black

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Stephen G Waxman

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P2860

Common molecular determinants of local anesthetic, antiarrhythmic, and anticonvulsant block of voltage-gated Na+ channels

Axonal transection in the lesions of multiple sclerosis

Hypoxia-like tissue injury as a component of multiple sclerosis lesions.

Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesions.

Noninactivating, tetrodotoxin-sensitive Na+ conductance in rat optic nerve axons.

Persistent functional deficit in multiple sclerosis and autosomal dominant cerebellar ataxia is associated with axon loss.

Phenytoin: mechanisms of its anticonvulsant action.

Reactive nitrogen intermediates in human neuropathology: an overview.

Prazosin suppresses development of axonal damage in rats inoculated for experimental allergic encephalomyelitis.

Nitric oxide toxicity in CNS white matter: an in vitro study using rat optic nerve.

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