Glutamate decreases mitochondrial size and movement in primary forebrain neurons. - Wikidata - wikimedia - TriplyDB

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

http://www.wikidata.org/entity/Q44566043

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The mechanism of Ca2+ -dependent regulation of kinesin-mediated mitochondrial motility Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration Cell signaling and mitochondrial dynamics: Implications for neuronal function and neurodegenerative disease Critical pathogenic events underlying progression of neurodegeneration in glaucoma The axonal transport of mitochondria Signaling to the microtubule cytoskeleton: an unconventional role for CaMKII Dopamine inhibits mitochondrial motility in hippocampal neurons Inner membrane fusion mediates spatial distribution of axonal mitochondria Mitochondrial transport in neurons: impact on synaptic homeostasis and neurodegeneration.The non-canonical Wnt/PKC pathway regulates mitochondrial dynamics through degradation of the arm-like domain-containing protein Alex3 Automated detection of whole-cell mitochondrial motility and its dependence on cytoarchitectural integrity Mitochondrial trafficking in neurons Glutamate excitotoxicity in neurons triggers mitochondrial and endoplasmic reticulum accumulation of Parkin, and, in the presence of N-acetyl cysteine, mitophagy Morphological and bioenergetic demands underlying the mitophagy in post-mitotic neurons: the pink-parkin pathway Excitotoxic insult results in a long-lasting activation of CaMKIIα and mitochondrial damage in living hippocampal neurons Mislocalization of neuronal mitochondria reveals regulation of Wallerian degeneration and NMNAT/WLD(S)-mediated axon protection independent of axonal mitochondria.Reversible phosphorylation of Drp1 by cyclic AMP-dependent protein kinase and calcineurin regulates mitochondrial fission and cell death Brain-derived neurotrophic factor (BDNF)-induced mitochondrial motility arrest and presynaptic docking contribute to BDNF-enhanced synaptic transmission.A calcineurin docking motif (LXVP) in dynamin-related protein 1 contributes to mitochondrial fragmentation and ischemic neuronal injury.Nitric oxide-induced mitochondrial fission is regulated by dynamin-related GTPases in neurons.Measurement of instantaneous velocity vectors of organelle transport: mitochondrial transport and bioenergetics in hippocampal neurons.Differential structural plasticity of corticostriatal and thalamostriatal axo-spinous synapses in MPTP-treated Parkinsonian monkeys.Mitochondrial motility and vascular smooth muscle proliferation.Loss of forebrain MTCH2 decreases mitochondria motility and calcium handling and impairs hippocampal-dependent cognitive functions.Mitochondrial morphogenesis, dendrite development, and synapse formation in cerebellum require both Bcl-w and the glutamate receptor delta2 Stimulation of glutamate receptors in cultured hippocampal neurons causes Ca2+-dependent mitochondrial contraction.Mitochondrial Aspects of Synaptic Dysfunction in Alzheimer's Disease.RyR2-Mediated Ca2+ Release and Mitochondrial ROS Generation Partake in the Synaptic Dysfunction Caused by Amyloid β Peptide Oligomers.The mitochondrial Ras-related GTPase Miro: views from inside and outside the metazoan kingdom.Evaluation of autophagy using mouse models of brain injury.Neuromodulation and mitochondrial transport: live imaging in hippocampal neurons over long durations.Axonal transport rate decreased at the onset of optic neuritis in EAE mice Beyond counts and shapes: studying pathology of dendritic spines in the context of the surrounding neuropil through serial section electron microscopy.Synaptic vesicle exocytosis in hippocampal synaptosomes correlates directly with total mitochondrial volume.Glutamate induces mitochondrial dynamic imbalance and autophagy activation: preventive effects of selenium.Cyclophilin D deficiency rescues axonal mitochondrial transport in Alzheimer's neurons The effects of NAD+ on apoptotic neuronal death and mitochondrial biogenesis and function after glutamate excitotoxicity.New model of action for mood stabilizers: phosphoproteome from rat pre-frontal cortex synaptoneurosomal preparations.Regulation of axonal mitochondrial transport and its impact on synaptic transmission Bioenergetics of the spinal cord in experimental autoimmune encephalitis of rats.

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P2860

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

http://www.wikidata.org/entity/Q44566043

description

2003 nî lūn-bûn

@nan

2003年の論文

@ja

2003年学术文章

@wuu

2003年学术文章

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2003年学术文章

@zh-cn

2003年学术文章

@zh-hans

2003年学术文章

@zh-my

2003年学术文章

@zh-sg

2003年學術文章

@yue

2003年學術文章

@zh-hant

name

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@en

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@nl

type

label

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@en

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@nl

prefLabel

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@en

Glutamate decreases mitochondrial size and movement in primary forebrain neurons.

@nl

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JNEUROSCI.23-21-07881.2003

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Journal of Neuroscience

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Glutamate decreases mitochondrial size and movement in primary forebrain neurons

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P2093

Anthony J Filiano

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Geraldine J Kress

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Gordon L Rintoul

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Ian J Reynolds

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Jacques B Brocard

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7881-7888

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10.1523/JNEUROSCI.23-21-07881.2003

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