Arginase reciprocally regulates nitric oxide synthase activity and contributes to endothelial dysfunction in aging blood vessels.
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Post-translational regulation of endothelial nitric oxide synthase in vascular endotheliumArginase activities and global arginine bioavailability in wild-type and ApoE-deficient mice: responses to high fat and high cholesterol dietsIncreased endothelial exocytosis and generation of endothelin-1 contributes to constriction of aged arteriesS-nitrosylation in cardiovascular signalingNitric oxide and peroxynitrite in health and diseaseNitric oxide synthases, S-nitrosylation and cardiovascular health: from molecular mechanisms to therapeutic opportunities (review)You're only as old as your arteries: translational strategies for preserving vascular endothelial function with agingNitric oxide synthases in heart failureAge-related changes in brain support cells: Implications for stroke severityArginase as a Critical Prooxidant Mediator in the Binomial Endothelial Dysfunction-AtherosclerosisProbing the Specificity Determinants of Amino Acid Recognition by Arginase † ‡Nitric oxide synthases: regulation and functionSelective endothelial overexpression of arginase II induces endothelial dysfunction and hypertension and enhances atherosclerosis in miceAging and vascular endothelial function in humansIncreased arginase II activity contributes to endothelial dysfunction through endothelial nitric oxide synthase uncoupling in aged miceIntravenous administration of piceatannol, an arginase inhibitor, improves endothelial dysfunction in aged miceArginase modulates myocardial contractility by a nitric oxide synthase 1-dependent mechanismDevelopmental changes in arginase expression and activity in the lungL-Arginine supplementation or arginase inhibition augments reflex cutaneous vasodilatation in aged human skin.Cold-induced cutaneous vasoconstriction is mediated by Rho kinase in vivo in human skin.Up-regulation of arginase activity contributes to attenuated reflex cutaneous vasodilatation in hypertensive humans.Rho kinase-mediated local cold-induced cutaneous vasoconstriction is augmented in aged human skin.Nitrergic response to cyclophosphamide treatment in blood and bone marrowVascular dysfunction in retinopathy-an emerging role for arginaseChronic oral administration of the arginase inhibitor 2(S)-amino-6-boronohexanoic acid (ABH) improves erectile function in aged rats.Vitamin D is a regulator of endothelial nitric oxide synthase and arterial stiffness in miceCopper amine oxidase 8 regulates arginine-dependent nitric oxide production in Arabidopsis thaliana.Thrombin induces endothelial arginase through AP-1 activation.Estrogen and oxidative stress: A novel mechanism that may increase the risk for cardiovascular disease in women.Ascorbic acid or L-arginine improves cutaneous microvascular function in chronic kidney disease.Piceatannol-3'-O-beta-D-glucopyranoside as an active component of rhubarb activates endothelial nitric oxide synthase through inhibition of arginase activity.The role of arginase and rho kinase in cardioprotection from remote ischemic perconditioning in non-diabetic and diabetic rat in vivo.Arterial aging and subclinical arterial disease are fundamentally intertwined at macroscopic and molecular levels.Mechanisms of vascular aging: new perspectives.Caloric restriction: powerful protection for the aging heart and vasculature.Immunosuppressive activity enhances central carbon metabolism and bioenergetics in myeloid-derived suppressor cells in vitro models.Local arginase inhibition during early reperfusion mediates cardioprotection via increased nitric oxide production.p38 Mitogen-activated protein kinase (MAPK) increases arginase activity and contributes to endothelial dysfunction in corpora cavernosa from angiotensin-II-treated miceEstrogen-induced contraction of coronary arteries is mediated by superoxide generated in vascular smooth muscle.Endothelial progenitor dysfunction in the pathogenesis of diabetic retinopathy: treatment concept to correct diabetes-associated deficits.
P2860
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P2860
Arginase reciprocally regulates nitric oxide synthase activity and contributes to endothelial dysfunction in aging blood vessels.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Arginase reciprocally regulate ...... nction in aging blood vessels.
@en
Arginase reciprocally regulate ...... nction in aging blood vessels.
@nl
type
label
Arginase reciprocally regulate ...... nction in aging blood vessels.
@en
Arginase reciprocally regulate ...... nction in aging blood vessels.
@nl
prefLabel
Arginase reciprocally regulate ...... nction in aging blood vessels.
@en
Arginase reciprocally regulate ...... nction in aging blood vessels.
@nl
P2093
P1433
P1476
Arginase reciprocally regulate ...... nction in aging blood vessels.
@en
P2093
Amy Cernetich
Artin A Shoukas
Dan E Berkowitz
Daniel Nyhan
Hunter C Champion
Joshua M Hare
Khalid M Minhas
Sean Burke
P304
P356
10.1161/01.CIR.0000092948.04444.C7
P407
P577
2003-09-29T00:00:00Z