Trisomy 21 causes changes in the circulating proteome indicative of chronic autoinflammation.

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Lifespan analysis of brain development, gene expression and behavioral phenotypes in the Ts1Cje, Ts65Dn and Dp(16)1/Yey mouse models of Down syndrome.

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Trisomy 21 causes changes in the circulating proteome indicative of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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Trisomy 21 causes changes in t ...... e of chronic autoinflammation.

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P1476

Trisomy 21 causes changes in t ...... ve of chronic autoinflammation

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Ahwan Pandey

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Angela L Rachubinski

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Donald Evans

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Francis Hickey

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Joaquin M Espinosa

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Keith P Smith

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Kristine Wolter-Warmerdam

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Neil Markham

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Thomas Blumenthal

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Thomas H Hraha

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P2860

Health Supervision for Children With Down Syndrome

Updated national birth prevalence estimates for selected birth defects in the United States, 2004-2006

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Differential expression of ZAP-70 and Syk protein tyrosine kinases, and the role of this family of protein tyrosine kinases in TCR signaling

A common mutation in the fibroblast growth factor receptor 1 gene in Pfeiffer syndrome

Riboflavin kinase couples TNF receptor 1 to NADPH oxidase

Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants

A Common Single Nucleotide Polymorphism in Endoplasmic Reticulum Aminopeptidase 2 Induces a Specificity Switch That Leads to Altered Antigen Processing

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SPAP2, an Ig family receptor containing both ITIMs and ITAMs

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s41598-017-13858-3

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scholarly article

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10.1038/S41598-017-13858-3

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English

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Kelly D Sullivan

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2017-11-01T00:00:00Z

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29093484

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5665944

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Trisomy 21 causes changes in the circulating proteome indicative of chronic autoinflammation.

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P2860

P2860

Trisomy 21 causes changes in the circulating proteome indicative of chronic autoinflammation.

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