Akt controls vascular smooth muscle cell proliferation in vitro and in vivo by delaying G1/S exit. - Wikidata - wikimedia - TriplyDB

Akt controls vascular smooth muscle cell proliferation in vitro and in vivo by delaying G1/S exit.

Akt controls vascular smooth muscle cell proliferation in vitro and in vivo by delaying G1/S exit.

http://www.wikidata.org/entity/Q44646999

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Akt isoforms in vascular disease The critical role of Akt in cardiovascular function Erythropoietin increases expression and function of vascular copper- and zinc-containing superoxide dismutase p21-Activated Kinase 4 Promotes Intimal Hyperplasia and Vascular Smooth Muscle Cells Proliferation during Superficial Femoral Artery Restenosis after Angioplasty.Decreased vascular lesion formation in mice with inducible endothelial-specific expression of protein kinase Akt The multifunctional Ca2+/calmodulin-dependent kinase II delta (CaMKIIdelta) controls neointima formation after carotid ligation and vascular smooth muscle cell proliferation through cell cycle regulation by p21.Cardiac progenitor cell commitment is inhibited by nuclear Akt expression.Akt activation induces hypertrophy without contractile phenotypic maturation in airway smooth muscle Decellularized tissue-engineered blood vessel as an arterial conduit FABP4 induces vascular smooth muscle cell proliferation and migration through a MAPK-dependent pathway.Leukotriene-C4 synthase, a critical enzyme in the activation of store-independent Orai1/Orai3 channels, is required for neointimal hyperplasia.Adventitial contributions of the extracellular signal-regulated kinase and Akt pathways to neointimal hyperplasia.Endovascular Gene Delivery from a Stent Platform: Gene- Eluting Stents.Smooth muscle cell signal transduction: implications of vascular biology for vascular surgeons.Dying tumor cells stimulate proliferation of living tumor cells via caspase-dependent protein kinase Cδ activation in pancreatic ductal adenocarcinoma TGF-β and Smad3 modulate PI3K/Akt signaling pathway in vascular smooth muscle cells Effects of Fluid Shear Stress on a Distinct Population of Vascular Smooth Muscle Cells.Caveolin-3 Promotes a Vascular Smooth Muscle Contractile Phenotype AKT2 confers protection against aortic aneurysms and dissections.Role of cellular bioenergetics in smooth muscle cell proliferation induced by platelet-derived growth factor.Nogo-B Receptor Modulates Pulmonary Artery Smooth Muscle Cell Function in Developing Lungs.Arterialization of a vein graft promotes cell cycle progression through Akt and p38 mitogen-activated protein kinase pathways: impact of the preparation procedure Dependence of proliferative vascular smooth muscle cells on CD98hc (4F2hc, SLC3A2)A Novel Approach against Vascular Intimal Hyperplasia Through the Suppression of Girdin.Insulin-like growth-factor-1-induced PKB signaling and Egr-1 expression is inhibited by curcumin in A-10 vascular smooth muscle cells.Naftopidil, a selective alpha-1 adrenoceptor antagonist, inhibits growth of human prostate cancer cells by G1 cell cycle arrest.Phospholipase Cγ1 Mediates Intima Formation Through Akt-Notch1 Signaling Independent of the Phospholipase Activity.Adenosine-5'-triphosphate up-regulates proliferation of human cardiac fibroblasts.Time and flow-dependent changes in the p27(kip1) gene network drive maladaptive vascular remodeling.Enhanced susceptibility of cyclin kinase inhibitor p21 knockout mice to high fat diet induced atherosclerosis.Extracellular ATP-induced proliferation of adventitial fibroblasts requires phosphoinositide 3-kinase, Akt, mammalian target of rapamycin, and p70 S6 kinase signaling pathways.Dicer generates a regulatory microRNA network in smooth muscle cells that limits neointima formation during vascular repair.Loss of Spry1 attenuates vascular smooth muscle proliferation by impairing mitogen-mediated changes in cell cycle regulatory circuits.Semaphorin-3E attenuates neointimal formation via suppressing VSMCs migration and proliferation.Connexin37 reduces smooth muscle cell proliferation and intimal hyperplasia in a mouse model of carotid artery ligation.Tauroursodeoxycholate (TUDCA) inhibits neointimal hyperplasia by suppression of ERK via PKCα-mediated MKP-1 induction.Niclosamide inhibits vascular smooth muscle cell proliferation and migration and attenuates neointimal hyperplasia in injured rat carotid arteries.

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P2860

Akt controls vascular smooth muscle cell proliferation in vitro and in vivo by delaying G1/S exit.

http://www.wikidata.org/entity/Q44646999

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

@en

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

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type

label

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

@en

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

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prefLabel

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

@en

Akt controls vascular smooth m ...... in vivo by delaying G1/S exit.

@nl

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01.RES.0000105086.31909.1B

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Circulation Research

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Akt controls vascular smooth m ...... in vivo by delaying G1/S exit

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P2093

Marco Castagna

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Matie Shou

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Matthew D Ringel

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Motoyasu Saji

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Richard Baffour

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Shmuel Fuchs

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Stephen E Epstein

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Timothy D Kinnaird

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Yi Fu Zhou

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1059-1065

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scholarly article

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10.1161/01.RES.0000105086.31909.1B

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11

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93

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Eugenio Stabile

Eugenio Stabile

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2003-11-06T00:00:00Z

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14605018

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