Epinephrine-induced hyperpolarization of islet cells without KATP channels.
about
Both G i and G o heterotrimeric G proteins are required to exert the full effect of norepinephrine on the beta-cell K ATP channel.Pharmacological stimulation and inhibition of insulin secretion in mouse islets lacking ATP-sensitive K+ channelsImpaired sympathoadrenal axis function contributes to enhanced insulin secretion in prediabetic obese ratsEvolving insights regarding mechanisms for the inhibition of insulin release by norepinephrine and heterotrimeric G proteins.Long lasting synchronization of calcium oscillations by cholinergic stimulation in isolated pancreatic islets.K(+) channels: function-structural overview.Hormonal inhibition of endocytosis: novel roles for noradrenaline and G protein G(z).Epinephrine-induced hyperpolarization of pancreatic islet cells is sensitive to PI3K-PDK1 signaling.Impaired glucagon secretory responses in mice lacking the type 1 sulfonylurea receptor.
P2860
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P2860
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
description
2003 nî lūn-bûn
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name
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@en
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@nl
type
label
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@en
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@nl
prefLabel
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@en
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@nl
P2093
P2860
P1476
Epinephrine-induced hyperpolarization of islet cells without KATP channels.
@en
P2093
Alvaro Muñoz
Andrea Sieg
Joseph Bryan
Lydia Aguilar-Bryan
Michael Buchenau
Mitsuhiro Nakazaki
Susanne Ullrich
P2860
P304
P356
10.1152/AJPENDO.00365.2003
P577
2003-11-12T00:00:00Z