Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation. - Wikidata - wikimedia - TriplyDB

Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation.

Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation.

http://www.wikidata.org/entity/Q44710589

about

Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis.Competitive control of independent programs of tumor necrosis factor receptor-induced cell death by TRADD and RIP1 Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation Many stimuli pull the necrotic trigger, an overview Ripped to death Autophagy and NF-kappaB: fight for fate Post-translational modifications as key regulators of TNF-induced necroptosis Peroxiredoxins and the Regulation of Cell Death Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs Execution of RIPK3-regulated necrosis RIPK-dependent necrosis and its regulation by caspases: a mystery in five acts Evidence-based selection of training compounds for use in the mechanism-based integrated prediction of drug-induced liver injury in man The role of necroptosis in neurosurgical diseases Tumor cell survival pathways activated by photodynamic therapy: a molecular basis for pharmacological inhibition strategies Necrostatin-1 rescues mice from lethal irradiation Mitochondrial Protein PGAM5 Regulates Mitophagic Protection against Cell Necroptosis A HIF-1 target, ATIA, protects cells from apoptosis by modulating the mitochondrial thioredoxin, TRX2 cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production Novel CHOP activator LGH00168 induces necroptosis in A549 human lung cancer cells via ROS-mediated ER stress and NF-κB inhibition Inhibitor of kappaB kinase beta regulates redox homeostasis by controlling the constitutive levels of glutathione Photodynamic therapy combined to cisplatin potentiates cell death responses of cervical cancer cells.Acute myeloid leukemia-targeted toxin activates both apoptotic and necroptotic death mechanisms TNFalpha-induced macrophage death via caspase-dependent and independent pathways.Retaining MKP1 expression and attenuating JNK-mediated apoptosis by RIP1 for cisplatin resistance through miR-940 inhibition TRAF2 plays a key, nonredundant role in LIGHT-lymphotoxin beta receptor signaling.Ebola virus glycoprotein directly triggers T lymphocyte death despite of the lack of infection.Biogenic selenium nanoparticles induce ROS-mediated necroptosis in PC-3 cancer cells through TNF activation.Necroptosis as an alternative form of programmed cell death.Jacalin Has Chemopreventive Effects on Colon Cancer Development.Stage-specific expression of TNFα regulates bad/bid-mediated apoptosis and RIP1/ROS-mediated secondary necrosis in Birnavirus-infected fish cells Distinct roles of basal steady-state and induced H-ferritin in tumor necrosis factor-induced death in L929 cells.The cytotoxicity and anticancer mechanisms of alterporriol L, a marine bianthraquinone, against MCF-7 human breast cancer cells.The molecular regulation of programmed necrotic cell injury.TNF-induced necroptosis and PARP-1-mediated necrosis represent distinct routes to programmed necrotic cell death IKK/NF-kappaB signaling: balancing life and death--a new approach to cancer therapy.Genetic analysis of the role of tumor necrosis factor receptors in functional outcome after traumatic brain injury in mice.Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis The complex interplay between autophagy, apoptosis, and necrotic signals promotes T-cell homeostasis.Alternative cell death mechanisms in development and beyond.Hematopoietic RIPK1 deficiency results in bone marrow failure caused by apoptosis and RIPK3-mediated necroptosis.

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Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation.

http://www.wikidata.org/entity/Q44710589

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2003 nî lūn-bûn

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2003年學術文章

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2003年學術文章

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name

Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Journal of Biological Chemistry

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Tumor necrosis factor-induced ...... e oxygen species accumulation.

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Gang Min Hur

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Jamie Hong Tran

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Qing-Feng Yang

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Sergei A Nedospasov

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Swati Choksi

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Yong Lin

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You Sun Kim

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Zheng-gang Liu

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10822-10828

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10.1074/JBC.M313141200

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279

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