The latent membrane protein 1 of Epstein-Barr virus establishes an antiviral state via induction of interferon-stimulated genes.
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The interferon response inhibits HIV particle production by induction of TRIM22IRF7: activation, regulation, modification and functionInterferon regulatory factor 4 is involved in Epstein-Barr virus-mediated transformation of human B lymphocytesThe viral etiology of AIDS-associated malignanciesDelayed polarization of mononuclear phagocyte transcriptional program by type I interferon isoformsOncostatin M Enhances the Antiviral Effects of Type I Interferon and Activates Immunostimulatory Functions in Liver Epithelial CellsDual functions of interferon regulatory factors 7C in Epstein-Barr virus-mediated transformation of human B lymphocytesViral transformation for production of personalized type I interferonsToll-like receptor 7 stimulates the expression of Epstein-Barr virus latent membrane protein 1Constitutively Active MAVS Inhibits HIV-1 Replication via Type I Interferon Secretion and Induction of HIV-1 Restriction Factors.TLR-TRIF pathway enhances the expression of KSHV replication and transcription activator.Induction of TRIM22 by IFN-γ Involves JAK and PC-PLC/PKC, but Not MAPKs and pI3K/Akt/mTOR Pathways.Attack, parry and riposte: molecular fencing between the innate immune system and human herpesviruses.Tumor Suppressor p53 Stimulates the Expression of Epstein-Barr Virus Latent Membrane Protein 1.p300, but not PCAF, collaborates with IRF-1 in stimulating TRIM22 expression independently of its histone acetyltransferase activity.Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein-Barr positive epithelial cells.Alpha/beta interferons regulate murine gammaherpesvirus latent gene expression and reactivation from latency.B30.2/SPRY domain in tripartite motif-containing 22 is essential for the formation of distinct nuclear bodies.Manipulation of the toll-like receptor 7 signaling pathway by Epstein-Barr virus.Epstein-Barr virus inhibits Kaposi's sarcoma-associated herpesvirus lytic replication in primary effusion lymphomas.The latent membrane protein 1 of Epstein-Barr virus (EBV) primes EBV latency cells for type I interferon production.Nuclear factor κB represses the expression of latent membrane protein 1 in Epstein-Barr virus transformed cells.Mouse gammaherpesvirus-68 infection acts as a rheostat to set the level of type I interferon signaling in primary macrophages.Innate immune modulation in EBV infection.Everyone comes from somewhere: systemic lupus erythematosus and Epstein-Barr virus induction of host interferon and humoral anti-Epstein-Barr nuclear antigen 1 immunity
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P2860
The latent membrane protein 1 of Epstein-Barr virus establishes an antiviral state via induction of interferon-stimulated genes.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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name
The latent membrane protein 1 ...... f interferon-stimulated genes.
@en
The latent membrane protein 1 ...... f interferon-stimulated genes.
@nl
type
label
The latent membrane protein 1 ...... f interferon-stimulated genes.
@en
The latent membrane protein 1 ...... f interferon-stimulated genes.
@nl
prefLabel
The latent membrane protein 1 ...... f interferon-stimulated genes.
@en
The latent membrane protein 1 ...... f interferon-stimulated genes.
@nl
P2093
P2860
P356
P1476
The latent membrane protein 1 ...... of interferon-stimulated genes
@en
P2093
Catherine Kotalik
Luwen Zhang
Subash C Das
P2860
P304
46335-46342
P356
10.1074/JBC.M403966200
P407
P50
P577
2004-08-20T00:00:00Z