about
Different types of retinal inhibition have distinct neurotransmitter release properties.Interneuron circuits tune inhibition in retinal bipolar cells.GABAC receptor-mediated inhibition in the retina.Early Retinal Neuronal Dysfunction in Diabetic Mice: Reduced Light-Evoked Inhibition Increases Rod Pathway Signaling.Light adaptation alters inner retinal inhibition to shape OFF retinal pathway signaling.Inhibition to retinal rod bipolar cells is regulated by light levelsSlow changes in Ca2(+) cause prolonged release from GABAergic retinal amacrine cells.Light adaptation alters the source of inhibition to the mouse retinal OFF pathway.Multiple pathways of inhibition shape bipolar cell responses in the retinaNonlinear interactions between excitatory and inhibitory retinal synapses control visual output.Differential effects of ethanol on GABA(A) and glycine receptor-mediated synaptic currents in brain stem motoneurons.Mechanisms for the modulation of native glycine receptor channels by ethanol.GABA(A), GABA(C) and glycine receptor-mediated inhibition differentially affects light-evoked signalling from mouse retinal rod bipolar cells.Dopamine D1 receptor activation contributes to light-adapted changes in retinal inhibition to rod bipolar cellsReceptor and Transmitter Release Properties Set the Time Course of Retinal InhibitionInhibitory components of retinal bipolar cell receptive fields are differentially modulated by dopamine D1 receptorsDopamine D1 receptor activation reduces local inner retinal inhibition to light-adapted levels
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description
hulumtuese
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wetenschapper
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հետազոտող
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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Erika D. Eggers
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P106
P1153
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P31
P496
0000-0003-1465-7284