Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
about
Aldosterone in the brainMineralocorticoid-induced sodium appetite and renal salt retention: evidence for common signaling and effector mechanismsHindlimb unloading results in increased predisposition to cardiac arrhythmias and alters left ventricular connexin 43 expression.Hindlimb unloading elicits anhedonia and sympathovagal imbalanceLocal Renin-Angiotensin system in the reproductive system.Central losartan attenuates increases in arterial pressure and expression of FosB/ΔFosB along the autonomic axis associated with chronic intermittent hypoxia.Hindbrain mineralocorticoid mechanisms on sodium appetite.Regulation of plasma vasopressin and renin activity in conscious hindlimb-unloaded rats.
P2860
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P2860
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
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Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
@en
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
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type
label
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
@en
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
@nl
prefLabel
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
@en
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading.
@nl
P2093
P2860
P1476
Rats exhibit aldosterone-dependent sodium appetite during 24 h hindlimb unloading
@en
P2093
Eileen M Hasser
Julia A Moffitt
Margaret J Sullivan
Stacy B Bruno
P2860
P304
P356
10.1113/JPHYSIOL.2004.062265
P407
P577
2004-03-26T00:00:00Z