Cardiac myocyte-specific HIF-1alpha deletion alters vascularization, energy availability, calcium flux, and contractility in the normoxic heart.
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Transcriptome and Molecular Endocrinology Aspects of Epicardial Adipose Tissue in Cardiovascular Diseases: A Systematic Review and Meta-Analysis of Observational StudiesHIF-1alpha Deficiency Attenuates the Cardiomyogenesis of Mouse Embryonic Stem CellsThe cell biology of disease: cellular mechanisms of cardiomyopathy.Hypoxia-inducible factor-dependent degeneration, failure, and malignant transformation of the heart in the absence of the von Hippel-Lindau proteinCardiomyocyte GATA4 functions as a stress-responsive regulator of angiogenesis in the murine heartOxygen, oxidative stress, hypoxia, and heart failure.Impaired Ca(2+)-handling in HIF-1alpha(+/-) mice as a consequence of pressure overload.The role of hypoxia inducible factor-1 in hepatocellular carcinoma.Cardiac-specific overexpression of HIF-1{alpha} prevents deterioration of glycolytic pathway and cardiac remodeling in streptozotocin-induced diabetic mice.Acute Vhl gene inactivation induces cardiac HIF-dependent erythropoietin gene expression.Hypoxia inducible factor 1 alpha regulates T cell receptor signal transduction.The heart: mostly postmitotic or mostly premitotic? Myocyte cell cycle, senescence, and quiescence.Redox signaling in cardiac myocytes.Cardiomyocyte-specific prolyl-4-hydroxylase domain 2 knock out protects from acute myocardial ischemic injuryHypoxia increases sirtuin 1 expression in a hypoxia-inducible factor-dependent manner.Cardiac failure in C5-deficient A/J mice after Candida albicans infectionPartial deficiency of HIF-1α stimulates pathological cardiac changes in streptozotocin-induced diabetic mice.Cardiac-specific ablation of ARNT leads to lipotoxicity and cardiomyopathy.HIF1α Represses Cell Stress Pathways to Allow Proliferation of Hypoxic Fetal Cardiomyocytes.Endothelial expression of hypoxia-inducible factor 1 protects the murine heart and aorta from pressure overload by suppression of TGF-β signaling.Heart-Specific Knockout of the Mitochondrial Thioredoxin Reductase (Txnrd2) Induces Metabolic and Contractile Dysfunction in the Aging Myocardium.Regression of pathological cardiac hypertrophy: signaling pathways and therapeutic targets.Normal glucose uptake in the brain and heart requires an endothelial cell-specific HIF-1α-dependent function.Engineered zinc-finger proteins can compensate genetic haploinsufficiency by transcriptional activation of the wild-type allele: application to Willams-Beuren syndrome and supravalvular aortic stenosisProtective effect of HDL on NADPH oxidase-derived super oxide anion mediates hypoxia-induced cardiomyocyte apoptosis.Cardiac responses to hypoxia and reoxygenation in Drosophila.Intravenous high mobility group box 1 upregulates the expression of HIF-1α in the myocardium via a protein kinase B-dependent pathway in rats following acute myocardial ischemia.The use of scaffold-free cell sheet technique to refine mesenchymal stromal cell-based therapy for heart failure.EGLN1 Inhibition and Rerouting of α-Ketoglutarate Suffice for Remote Ischemic Protection.MicroRNA-210 decreases heme levels by targeting ferrochelatase in cardiomyocytes.Endothelial Nogo-B regulates sphingolipid biosynthesis to promote pathological cardiac hypertrophy during chronic pressure overload.AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury.Delineation of Molecular Pathways Involved in Cardiomyopathies Caused by Troponin T MutationsRedox signaling in cardiovascular health and disease.The von Hippel-Lindau Chuvash mutation in mice alters cardiac substrate and high-energy phosphate metabolism.Mitochondria and energetic depression in cell pathophysiologyHypoxia inducible factor pathway inhibitors as anticancer therapeutics.Network-based association of hypoxia-responsive genes with cardiovascular diseasesA designed zinc-finger transcriptional repressor of phospholamban improves function of the failing heart.Oxygen tension regulates preosteocyte maturation and mineralization.
P2860
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P2860
Cardiac myocyte-specific HIF-1alpha deletion alters vascularization, energy availability, calcium flux, and contractility in the normoxic heart.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@en
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@nl
type
label
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@en
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@nl
prefLabel
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@en
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@nl
P2093
P356
P1433
P1476
Cardiac myocyte-specific HIF-1 ...... ctility in the normoxic heart.
@en
P2093
Agnes Dadak
Dinggang Liu
Frank J Giordano
Jennifer L Yeh
Lawrence H Young
Reed P Hickey
P304
P356
10.1096/FJ.04-1510FJE
P407
P577
2004-05-07T00:00:00Z