Ex vivo zidovudine (AZT) treatment of CD34+ bone marrow progenitors causes decreased steady state mitochondrial DNA (mtDNA) and increased lactate production.
about
Neurotoxic effects of AZT on developing and adult neurogenesisOxazolidinones inhibit cellular proliferation via inhibition of mitochondrial protein synthesisHematological Indices at Birth of Infants of HIV-Positive Mothers Participating in a Prevention of Mother-to-Child Transmission ProgramHIV-exposed uninfected children: a growing population with a vulnerable immune system?Influence of new antiretrovirals on hematological toxicity in HIV-exposed uninfected infants.Vitamins C and E prevent AZT-induced leukopenia and loss of cellularity in bone marrow. Studies in mice.
P2860
Ex vivo zidovudine (AZT) treatment of CD34+ bone marrow progenitors causes decreased steady state mitochondrial DNA (mtDNA) and increased lactate production.
description
2004 nî lūn-bûn
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2004年の論文
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name
Ex vivo zidovudine (AZT) treat ...... increased lactate production.
@en
Ex vivo zidovudine
@nl
type
label
Ex vivo zidovudine (AZT) treat ...... increased lactate production.
@en
Ex vivo zidovudine
@nl
prefLabel
Ex vivo zidovudine (AZT) treat ...... increased lactate production.
@en
Ex vivo zidovudine
@nl
P2093
P2860
P1476
Ex vivo zidovudine (AZT) treat ...... increased lactate production.
@en
P2093
P2860
P304
P356
10.1191/0960327104HT437OA
P577
2004-04-01T00:00:00Z