Bacteria challenge in smoke-exposed mice exacerbates inflammation and skews the inflammatory profile.
about
Lung microbiology and exacerbations in COPDNeutrophil Extracellular Traps in Pulmonary Diseases: Too Much of a Good Thing?IL-1α/IL-1R1 expression in chronic obstructive pulmonary disease and mechanistic relevance to smoke-induced neutrophilia in miceNrf2 regulates chronic lung inflammation and B-cell responses to nontypeable Haemophilus influenzaeDivergent pro-inflammatory profile of human dendritic cells in response to commensal and pathogenic bacteria associated with the airway microbiotaTreating viral exacerbations of chronic obstructive pulmonary disease: insights from a mouse model of cigarette smoke and H1N1 influenza infectionWhat drives the peripheral lung-remodeling process in chronic obstructive pulmonary disease?Impact of inflammation, emphysema, and smoking cessation on V/Q in mouse models of lung obstruction.New aspects on the structure of neutrophil extracellular traps from chronic obstructive pulmonary disease and in vitro generation.Cigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.The role of the CCR1 receptor in the inflammatory response to tobacco smoke in a mouse modelExposing rodents to a combination of tobacco smoke and lipopolysaccharide results in an exaggerated inflammatory response in the lung.Inhibition by cigarette smoke of nuclear factor-κB-dependent response to bacteria in the airway.Nontypeable Haemophilus influenzae induces sustained lung oxidative stress and protease expression.Cigarette smoke-promoted acquisition of bacterial pathogens in the upper respiratory tract leads to enhanced inflammation in mice.Neutrophil extracellular trap (NET) formation characterises stable and exacerbated COPD and correlates with airflow limitationThe role of the local microbial ecosystem in respiratory health and disease.Elastase/LPS-exposed mice exhibit impaired innate immune responses to bacterial challenge: role of scavenger receptor A.The role of TLR2 and bacterial lipoprotein in enhancing airway inflammation and immunityNeural signaling in the spleen controls B-cell responses to blood-borne antigen.IL-22 Defect During Streptococcus pneumoniae Infection Triggers Exacerbation of Chronic Obstructive Pulmonary DiseaseAlcohol abuse and smoking alter inflammatory mediator production by pulmonary and systemic immune cells.NTHi induction of Cxcl2 and middle ear mucosal metaplasia in mice.An in vitro model of murine middle ear epithelium.Update in chronic obstructive pulmonary disease 2009.The Lung Immune Response to Nontypeable Haemophilus influenzae (Lung Immunity to NTHi).Genome Expression Profiling-Based Identification and Administration Efficacy of Host-Directed Antimicrobial Drugs against Respiratory Infection by Nontypeable Haemophilus influenzae.CD4+CD25+FoxP3+ Regulatory Tregs inhibit fibrocyte recruitment and fibrosis via suppression of FGF-9 production in the TGF-β1 exposed murine lung.Interleukin-22 protects against non-typeable Haemophilus influenzae infection: alteration during chronic obstructive pulmonary disease.IL-17A and the Promotion of Neutrophilia in Acute Exacerbation of Chronic Obstructive Pulmonary Disease.Chronic obstructive pulmonary disease and asthma-associated Proteobacteria, but not commensal Prevotella spp., promote Toll-like receptor 2-independent lung inflammation and pathology.Combined exposure to cigarette smoke and nontypeable Haemophilus influenzae drives development of a COPD phenotype in mice.A single dose of lipopolysaccharide into mice with emphysema mimics human chronic obstructive pulmonary disease exacerbation as assessed by micro-computed tomography.Global state of tobacco use: summary from the American Thoracic Society International Conference 2016.Resveratrol therapeutics combines both antimicrobial and immunomodulatory properties against respiratory infection by nontypeable Haemophilus influenzae.Exacerbation of cigarette smoke-induced pulmonary inflammation by Staphylococcus aureus enterotoxin B in mice.Haemophilus influenzae and the lung (Haemophilus and the lung).Cigarette smoke exposure as a model of inflammation associated with COPD.Bone marrow mesenchymal stem cells ameliorate lung injury through anti-inflammatory and antibacterial effect in COPD mice.Are lymphoid follicles important in the pathogenesis of chronic obstructive pulmonary disease?
P2860
Q22305860-0E45A6E8-E52A-4B04-A197-957C1A9904FBQ26738321-BBAFA063-308C-444E-BE07-33A429CB1745Q27311393-43611782-52D3-47AE-8F37-D156F2910BCFQ28392051-247600F2-7E10-47C9-97D2-EE2BA951C3CFQ28731877-87B89DA8-3A49-420C-9EC2-F065909535EAQ28748904-FA11DCE1-F54B-4078-8D80-E482ED3EE79DQ33518335-A13706E5-EAE8-44A5-A453-7AB2499B2BA4Q33611889-E473CF7A-3417-4C95-A5EB-97FB6FB6B110Q33618491-8BB293C0-21B2-4738-AC41-CD3E0FAA73DCQ33790352-899CDD93-88E5-4D0E-9E7B-B08BC6FC77BCQ34106030-EC6859C5-45EC-46A4-933F-F284B5E80D13Q34219585-17EB44EE-45E8-4984-A9B3-C2EC7312B345Q34626521-BD0B29F3-766B-4E52-AB22-2A32A38A8121Q35203322-7773E26E-AE66-4342-AB71-E9B25F1DF361Q35400725-154B5BE9-A679-4EB9-87FE-0D1099B099D0Q35637182-DA13E627-6506-4659-B5CE-7813CDD8D4AAQ35684946-5C442D48-A294-4626-B982-E0F1EFEA2458Q35917701-802463BD-F31E-4B3C-B033-66E74D8185A0Q35927593-D3CF83D9-D63B-453C-80DA-533A061E02B4Q36071721-F631145C-EE4C-430B-87C9-7C6431F786CFQ36540978-05CB987B-BD5E-4795-A2B6-92A92A9992C6Q36698703-37B9E61C-0FCB-4D0D-94BA-D1B6CD5E0656Q37086318-351A1818-A58C-4F55-8CAF-39CACA0C8966Q37425982-3BF0882E-9F8E-4320-9093-256B97A7FFD4Q37718404-A90AADCE-76E4-4FDA-A44C-3DFA9F0B9973Q38538384-0F4E2A73-D7E2-4A91-B565-0CFEB2B46498Q38831662-9A727529-1F26-4F7C-B0C9-0D0AD0BCBD5CQ39919706-B34F9D53-7317-46AD-A257-C99EA4C675E2Q40688717-208A6937-C811-44F5-B981-BAAE7BAE7082Q41187761-A0DECE13-569B-4231-A35A-A4276F579AFEQ41758607-9882563B-F746-49DC-90F8-5D9D056FF762Q41959561-DAE7C667-DB62-4922-B9C6-617215BB65B7Q42286333-0406D4B1-BA26-48C7-9B4F-D0E27C5EE2EEQ42346107-53B726F7-92E6-4E4E-8B50-FB0E72F9945EQ42379026-4A73BD22-D467-4142-ACE6-4990959B1405Q42777927-9FA44A82-ADB6-40E2-A6E5-0FA97EE9103FQ43212173-59D08ACC-F401-4263-B63C-E6B510D42399Q46328869-00074754-C401-40D5-A0EB-2C4B04522B35Q47808641-ED5BE27A-0A74-458F-A40E-9AA1D5F49BA0Q47885104-2682E8D8-7D09-410E-A3F8-81B34639C363
P2860
Bacteria challenge in smoke-exposed mice exacerbates inflammation and skews the inflammatory profile.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@en
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@nl
type
label
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@en
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@nl
prefLabel
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@en
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@nl
P2093
P2860
P1476
Bacteria challenge in smoke-ex ...... kews the inflammatory profile.
@en
P2093
Caleb C J Zavitz
Gordon J Gaschler
Maria Lindahl
Marko Skrtic
Martin R Stämpfli
Per-Ola Onnervik
Sanjay Sethi
P2860
P304
P356
10.1164/RCCM.200808-1306OC
P407
P577
2009-01-29T00:00:00Z