Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
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Cellular demise and inflammatory microglial activation during beta-amyloid toxicity are governed by Wnt1 and canonical signaling pathways.Prevention of β-amyloid degeneration of microglia by erythropoietin depends on Wnt1, the PI 3-K/mTOR pathway, Bad, and Bcl-xL.Triple play: promoting neurovascular longevity with nicotinamide, WNT, and erythropoietin in diabetes mellitus.The Wnt signaling pathway: aging gracefully as a protectionist?It all starts at the ends: multifaceted involvement of C- and N-terminally modified cholinesterases in Alzheimer's disease.
P2860
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
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2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@en
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@nl
type
label
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@en
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@nl
prefLabel
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@en
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@nl
P2093
P1476
Wnt-1 expression in PC12 cells induces exon 15 deletion and expression of L-APP.
@en
P2093
Anthony M C Brown
Kenneth S Kosik
Mikhail Semenov
Peter J Morin
P356
10.1016/J.NBD.2004.01.004
P577
2004-06-01T00:00:00Z