Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus. - Wikidata - wikimedia - TriplyDB

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

http://www.wikidata.org/entity/Q44945685

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Spike timing-dependent synaptic depression in the in vivo barrel cortex of the rat Tonotopic reorganization of developing auditory brainstem circuits Maladaptive plasticity in tinnitus--triggers, mechanisms and treatment.The spike-timing dependence of plasticity ON and OFF unipolar brush cells transform multisensory inputs to the auditory system.Saturation of long-term potentiation in the dorsal cochlear nucleus and its pharmacological reversal in an experimental model of tinnitus The Contribution of Brainstem and Cerebellar Pathways to Auditory Recognition.Tinnitus: Maladaptive auditory-somatosensory plasticity.Bidirectional Shifting Effects of the Sound Intensity on the Best Frequency in the Rat Auditory Cortex.Bimodal stimulus timing-dependent plasticity in primary auditory cortex is altered after noise exposure with and without tinnitus.β-Arrestin-Dependent Dopaminergic Regulation of Calcium Channel Activity in the Axon Initial Segment.Tonic zinc inhibits spontaneous firing in dorsal cochlear nucleus principal neurons by enhancing glycinergic neurotransmission.Increased Synchrony and Bursting of Dorsal Cochlear Nucleus Fusiform Cells Correlate with Tinnitus.Listening to another sense: somatosensory integration in the auditory system AMPA receptor inhibition by synaptically released zinc Stimulus-timing-dependent modifications of rate-level functions in animals with and without tinnitus.Induction of motor associative plasticity in the posterior parietal cortex-primary motor network Potent KCNQ2/3-specific channel activator suppresses in vivo epileptic activity and prevents the development of tinnitus Suppression and facilitation of auditory neurons through coordinated acoustic and midbrain stimulation: investigating a deep brain stimulator for tinnitus NMDA Receptors Mediate Stimulus-Timing-Dependent Plasticity and Neural Synchrony in the Dorsal Cochlear Nucleus.Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus.Serotonergic regulation of excitability of principal cells of the dorsal cochlear nucleus.Transcutaneous induction of stimulus-timing-dependent plasticity in dorsal cochlear nucleus.Tinnitus: animal models and findings in humans.Chemical synaptic transmission onto superficial stellate cells of the mouse dorsal cochlear nucleus.Investigating a new neuromodulation treatment for brain disorders using synchronized activation of multimodal pathways.Cell-type specific short-term plasticity at auditory nerve synapses controls feed-forward inhibition in the dorsal cochlear nucleus.Modulation of electrocortical brain activity by attention in individuals with and without tinnitus.Stimulus timing-dependent plasticity in dorsal cochlear nucleus is altered in tinnitus.Regulation of interneuron excitability by gap junction coupling with principal cells.Synaptic Zn2+ inhibits neurotransmitter release by promoting endocannabinoid synthesis.Intrinsic and synaptic properties of vertical cells of the mouse dorsal cochlear nucleus.Diverse levels of an inwardly rectifying potassium conductance generate heterogeneous neuronal behavior in a population of dorsal cochlear nucleus pyramidal neurons.Stimulus-timing dependent multisensory plasticity in the guinea pig dorsal cochlear nucleus.Noise overexposure alters long-term somatosensory-auditory processing in the dorsal cochlear nucleus--possible basis for tinnitus-related hyperactivity?Plasticity of somatosensory inputs to the cochlear nucleus--implications for tinnitus Development of auditory cortical synaptic receptive fields.Mechanisms underlying input-specific expression of endocannabinoid-mediated synaptic plasticity in the dorsal cochlear nucleus Spectrotemporal dynamics of auditory cortical synaptic receptive field plasticity.Spontaneous spiking and synaptic depression underlie noradrenergic control of feed-forward inhibition

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P2860

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

http://www.wikidata.org/entity/Q44945685

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

@zh-hant

name

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@en

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@nl

type

label

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@en

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@nl

prefLabel

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@en

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus.

@nl

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Nature Neuroscience

P1476

Cell-specific, spike timing-dependent plasticities in the dorsal cochlear nucleus

@en

P2093

Laurence O Trussell

http://www.w3.org/2001/XMLSchema#string

Thanos Tzounopoulos

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P2860

Regulation of synaptic efficacy by coincidence of postsynaptic APs and EPSPs

Bidirectional synaptic plasticity in the cerebellum-like mammalian dorsal cochlear nucleus.

Timing-based LTP and LTD at vertical inputs to layer II/III pyramidal cells in rat barrel cortex.

Rate, timing, and cooperativity jointly determine cortical synaptic plasticity.

Evolution of cerebellum-like structures.

Equalization of synaptic efficacy by activity- and timing-dependent synaptic plasticity.

A mechanism for the Hebb and the anti-Hebb processes underlying learning and memory.

Spike timing, calcium signals and synaptic plasticity.

Distribution and targets of the cartwheel cell axon in the dorsal cochlear nucleus of the guinea pig.

What's a cerebellar circuit doing in the auditory system?

P2888

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719-725

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scholarly article

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10.1038/NN1272

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7

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7

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2004-06-20T00:00:00Z

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8498779

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15208632

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