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Compromised mitochondrial fatty acid synthesis in transgenic mice results in defective protein lipoylation and energy disequilibriumGalectin-1 Protein Therapy Prevents Pathology and Improves Muscle Function in the mdx Mouse Model of Duchenne Muscular DystrophyArginine metabolism by macrophages promotes cardiac and muscle fibrosis in mdx muscular dystrophy.Exon skipping restores dystrophin expression, but fails to prevent disease progression in later stage dystrophic dko miceThe effects of glucocorticoid and voluntary exercise treatment on the development of thoracolumbar kyphosis in dystrophin-deficient mice.A farnesyltransferase inhibitor improves disease phenotypes in mice with a Hutchinson-Gilford progeria syndrome mutation.Ventilatory chemosensory drive is blunted in the mdx mouse model of Duchenne Muscular Dystrophy (DMD).Neurofibromin (Nf1) is required for skeletal muscle developmentSERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models.Severe Extracellular Matrix Abnormalities and Chondrodysplasia in Mice Lacking Collagen Prolyl 4-Hydroxylase Isoenzyme II in Combination with a Reduced Amount of Isoenzyme I.Dystrophin and utrophin expression require sarcospan: loss of α7 integrin exacerbates a newly discovered muscle phenotype in sarcospan-null mice.The role of Sema3-Npn-1 signaling during diaphragm innervation and muscle development.Removal of immunoglobulin-like domains from titin's spring segment alters titin splicing in mouse skeletal muscle and causes myopathy.The paradox of muscle hypertrophy in muscular dystrophy.Cardiac and respiratory dysfunction in Duchenne muscular dystrophy and the role of second messengers.What do mouse models of muscular dystrophy tell us about the DAPC and its components?Muscleblind-like 2 (Mbnl2) -deficient mice as a model for myotonic dystrophy.Plantarflexion contracture in the mdx mouse.Systemic AAV-Mediated β-Sarcoglycan Delivery Targeting Cardiac and Skeletal Muscle Ameliorates Histological and Functional Deficits in LGMD2E Mice.Human α7 Integrin Gene (ITGA7) Delivered by Adeno-Associated Virus Extends Survival of Severely Affected Dystrophin/Utrophin-Deficient MiceVentilation during air breathing and in response to hypercapnia in 5 and 16 month-old mdx and C57 mice.Tauopathy induced by low level expression of a human brain-derived tau fragment in mice is rescued by phenylbutyrate.Treatment with the anti-IL-6 receptor antibody attenuates muscular dystrophy via promoting skeletal muscle regeneration in dystrophin-/utrophin-deficient mice.Congenital muscle dystrophy and diet consistency affect mouse skull shape differently.Skeletal Characterization of the Fgfr3 Mouse Model of Achondroplasia Using Micro-CT and MRI Volumetric Imaging.How to Wire the Diaphragm: Wholemount Staining Methods to Analyze Mammalian Respiratory Innervation.Quantitation of sleep and spinal curvature in an unusually longevous owl monkey (Aotus azarae).Targeting of NAT10 enhances healthspan in a mouse model of human accelerated aging syndrome.Chronic Paraspinal Muscle Injury Model in Rat.Chronic Paraspinal Muscle Injury Model in Rat.Postnatal soluble FGFR3 therapy rescues achondroplasia symptoms and restores bone growth in mice.A cell autonomous torsinA requirement for cholinergic neuron survival and motor control
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Progression of kyphosis in mdx mice.
@en
Progression of kyphosis in mdx mice.
@nl
type
label
Progression of kyphosis in mdx mice.
@en
Progression of kyphosis in mdx mice.
@nl
prefLabel
Progression of kyphosis in mdx mice.
@en
Progression of kyphosis in mdx mice.
@nl
P2860
P1476
Progression of kyphosis in mdx mice.
@en
P2093
Andrew Hoey
Nicola Laws
P2860
P304
P356
10.1152/JAPPLPHYSIOL.01357.2003
P407
P577
2004-07-02T00:00:00Z