Evaluation of the mechanism underlying the inhibitory effect of guanidinoacetate on brain Na+, K+-ATPase activity.
about
Guanidinoacetic acid as a performance-enhancing agent.Creatine synthesis and exchanges between brain cells: What can be learned from human creatine deficiencies and various experimental models?Creatine prevents the inhibition of energy metabolism and lipid peroxidation in rats subjected to GAA administration.Alcohol consumption decreases rat hepatic creatine biosynthesis via altered guanidinoacetate methyltransferase activity.3-Methylcrotonylglycine disrupts mitochondrial energy homeostasis and inhibits synaptic Na(+),K (+)-ATPase activity in brain of young rats.Defining the pathogenicity of creatine deficiency syndrome.
P2860
Evaluation of the mechanism underlying the inhibitory effect of guanidinoacetate on brain Na+, K+-ATPase activity.
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2004 nî lūn-bûn
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name
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@en
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@nl
type
label
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@en
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@nl
prefLabel
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@en
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity.
@nl
P2093
P1476
Evaluation of the mechanism un ...... brain Na+, K+-ATPase activity
@en
P2093
Alexandra I Zugno
Clovis M D Wannmacher
Fábria Chiarani
Moacir Wajner
Renata Franzon
P304
P356
10.1016/J.IJDEVNEU.2004.05.002
P577
2004-06-01T00:00:00Z