CXCR2 is essential for maximal neutrophil recruitment and methacholine responsiveness after ozone exposure.
about
Inflammasome, IL-1 and inflammation in ozone-induced lung injuryChemokine receptors and their therapeutic opportunities in diseased lung: far beyond leukocyte traffickingGenes of innate immunity and the biological response to inhaled ozoneTargeting CXCR1/CXCR2 receptor antagonism in malignant melanomaGenetic Ablation of CXCR2 Protects against Cigarette Smoke-Induced Lung Inflammation and InjuryCritical role of endothelial CXCR2 in LPS-induced neutrophil migration into the lung.TLR4 is necessary for hyaluronan-mediated airway hyperresponsiveness after ozone inhalation.Onset of obesity in carboxypeptidase E-deficient mice and effect on airway responsiveness and pulmonary responses to ozoneImpact of adiponectin deficiency on pulmonary responses to acute ozone exposure in mice.CRTH2 is a critical regulator of neutrophil migration and resistance to polymicrobial sepsis.Evaluating potential response-modifying factors for associations between ozone and health outcomes: a weight-of-evidence approach.The role of the extracellular matrix protein mindin in airway response to environmental airways injuryMFG-E8 inhibits neutrophil migration through αvβ₃-integrin-dependent MAP kinase activation.Pulmonary inflammation induced by subacute ozone is augmented in adiponectin-deficient mice: role of IL-17A.Impact of aging on pulmonary responses to acute ozone exposure in mice: role of TNFR1.ROCK insufficiency attenuates ozone-induced airway hyperresponsiveness in miceProtective role of matrix metalloproteinase-9 in ozone-induced airway inflammation.CXCR2-dependent mucosal neutrophil influx protects against colitis-associated diarrhea caused by an attaching/effacing lesion-forming bacterial pathogen.Resistin deficiency in mice has no effect on pulmonary responses induced by acute ozone exposure.Pulmonary CXCR2 regulates VCAM-1 and antigen-induced recruitment of mast cell progenitorsType I interleukin-1 receptor is required for pulmonary responses to subacute ozone exposure in mice.Neutrophils Oppose Uterine Epithelial Carcinogenesis via Debridement of Hypoxic Tumor Cells.Facilitation of Allergic Sensitization and Allergic Airway Inflammation by Pollen-Induced Innate Neutrophil Recruitment.Obesity and asthma: lessons from animal models.Role of CXCR2 in cigarette smoke-induced lung inflammation.Ozone and pulmonary innate immunityAugmented pulmonary responses to acute ozone exposure in obese mice: roles of TNFR2 and IL-13.No effect of metformin on the innate airway hyperresponsiveness and increased responses to ozone observed in obese miceEndogenous osteopontin promotes ozone-induced neutrophil recruitment to the lungs and airway hyperresponsiveness to methacholineAmbient ozone and pulmonary innate immunity.Host CXCR2-dependent regulation of melanoma growth, angiogenesis, and experimental lung metastasis.Metallothionein as an anti-inflammatory mediatorPlasminogen activator inhibitor-1 does not contribute to the pulmonary pathology induced by acute exposure to ozone.Lung NF-kappaB activation and neutrophil recruitment require IL-1 and TNF receptor signaling during pneumococcal pneumonia.The functional significance behind expressing two IL-8 receptor types on PMN.IL-33 Drives Augmented Responses to Ozone in Obese Mice.Neutrophils in local and systemic antibody-dependent inflammatory and anaphylactic reactions.Blockade of CXCR2 signalling: a potential therapeutic target for preventing neutrophil-mediated inflammatory diseases.Increased pulmonary responses to acute ozone exposure in obese db/db mice.Augmented responses to ozone in obese carboxypeptidase E-deficient mice.
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P2860
CXCR2 is essential for maximal neutrophil recruitment and methacholine responsiveness after ozone exposure.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@en
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@nl
type
label
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@en
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@nl
prefLabel
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@en
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@nl
P2860
P1476
CXCR2 is essential for maximal ...... siveness after ozone exposure.
@en
P2093
Richard A Johnston
Stephanie A Shore
P2860
P356
10.1152/AJPLUNG.00101.2004
P577
2004-09-10T00:00:00Z