Ischemia-reperfusion induces glomerular and tubular activation of proinflammatory and antiapoptotic pathways: differential modulation by rapamycin.
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Effect of sulfasalazine on renal ischemia/reperfusion injury in ratsTWEAK activates the non-canonical NFkappaB pathway in murine renal tubular cells: modulation of CCL21B-cell CLL/lymphoma 10 (BCL10) is required for NF-kappaB production by both canonical and noncanonical pathways and for NF-kappaB-inducing kinase (NIK) phosphorylation.Rapamycin protection of livers from ischemia and reperfusion injury is dependent on both autophagy induction and mammalian target of rapamycin complex 2-Akt activation.Karyopherins: potential biological elements involved in the delayed graft function in renal transplant recipients.NF-κB-inducing kinase increases renal tubule epithelial inflammation associated with diabetesHuR inhibits apoptosis by amplifying Akt signaling through a positive feedback loop.Mycophenolate mofetil and sirolimus combination in renal transplantation.Immune and inflammatory role in renal disease.Mammalian target of rapamycin and the kidney. II. Pathophysiology and therapeutic implications.TWEAK/Fn14 and Non-Canonical NF-kappaB Signaling in Kidney DiseaseGenomic profiling of kidney ischemia-reperfusion reveals expression of specific alloimmunity-associated genes: Linking "immune" and "nonimmune" injury events.Roles of mTOR complexes in the kidney: implications for renal disease and transplantation.Mitogen-Activated Protein Kinase 14 Promotes AKI.Methotrexate-loaded chitosan- and glycol chitosan-based nanoparticles: a promising strategy for the administration of the anticancer drug to brain tumors.Thrombin may modulate dendritic cell activation in kidney transplant recipients with delayed graft function.Incipient renal transplant dysfunction associates with tubular syndecan-1 expression and shedding.Bcl3: a regulator of NF-κB inducible by TWEAK in acute kidney injury with anti-inflammatory and antiapoptotic properties in tubular cells.Angiotensin II activation of mTOR results in tubulointerstitial fibrosis through loss of N-cadherin.Endothelial-to-mesenchymal transition and renal fibrosis in ischaemia/reperfusion injury are mediated by complement anaphylatoxins and Akt pathway.T helper 1, 2 and 17 cell subsets in renal transplant patients with delayed graft function.CD1d-associated expression of NF-kB and cardiac dysfunction in diabetic and obese mice.Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation.Lymphocyte-specific deletion of IKK2 or NEMO mediates an increase in intrarenal Th17 cells and accelerates renal damage in an ischemia-reperfusion injury mouse model.Up-regulation of cell cycle regulatory genes after renal ischemia/reperfusion: differential expression of p16(INK4a), p21(WAF1/CIP1) and p27(Kip1) cyclin-dependent kinase inhibitor genes depending on reperfusion time.
P2860
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P2860
Ischemia-reperfusion induces glomerular and tubular activation of proinflammatory and antiapoptotic pathways: differential modulation by rapamycin.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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2004年學術文章
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name
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@en
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@nl
type
label
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@en
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@nl
prefLabel
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@en
Ischemia-reperfusion induces g ...... ntial modulation by rapamycin.
@nl
P2093
P50
P1476
Ischemia-reperfusion induces g ...... ential modulation by rapamycin
@en
P2093
Antonia Loverre
Antonio Schena
Barbara Infante
Carmen Capobianco
Elena Ranieri
Francesco Paolo Schena
Francesco Paolo Selvaggi
Giuseppe Grandaliano
Loreto Gesualdo
Michele Battaglia
P304
P356
10.1097/01.ASN.0000139932.00971.E4
P577
2004-10-01T00:00:00Z