Long-term potentiation deficits and excitability changes following traumatic brain injury.
about
Making Waves in the Brain: What Are Oscillations, and Why Modulating Them Makes Sense for Brain InjuryRole of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute NeuropathologiesMolecular mechanisms of cognitive dysfunction following traumatic brain injuryPersistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesisDiffuse brain injury elevates tonic glutamate levels and potassium-evoked glutamate release in discrete brain regions at two days post-injury: an enzyme-based microelectrode array study.Select non-coding RNA in blood components provide novel clinically accessible biological surrogates for improved identification of traumatic brain injury in OEF/OIF Veterans.Decreased level of olfactory receptors in blood cells following traumatic brain injury and potential association with tauopathyModels of brain injury and alterations in synaptic plasticity.α-Synuclein levels are elevated in cerebrospinal fluid following traumatic brain injury in infants and children: the effect of therapeutic hypothermia.Astrocyte-Specific Overexpression of Insulin-Like Growth Factor-1 Protects Hippocampal Neurons and Reduces Behavioral Deficits following Traumatic Brain Injury in Mice.Genetic activation of mTORC1 signaling worsens neurocognitive outcome after traumatic brain injury.Excitatory synaptic transmission and network activity are depressed following mechanical injury in cortical neuronsTraumatic brain injury and its effects on synaptic plasticity.From cell death to neuronal regeneration: building a new brain after traumatic brain injury.Reliability of VEP Recordings Using Chronically Implanted Screw Electrodes in MiceAltered calcium signaling following traumatic brain injuryMapping the Connectome Following Traumatic Brain Injury.Decoding hippocampal signaling deficits after traumatic brain injuryTransition from Initial Hypoactivity to Hyperactivity in Cortical Layer V Pyramidal Neurons after Traumatic Brain Injury In Vivo.Elevated plasma MCP-1 concentration following traumatic brain injury as a potential "predisposition" factor associated with an increased risk for subsequent development of Alzheimer's disease.Traumatic axonal injury in the mouse is accompanied by a dynamic inflammatory response, astroglial reactivity and complex behavioral changesPhosphodiesterase inhibition rescues chronic cognitive deficits induced by traumatic brain injuryNeurotrophin-mediated neuroprotection of hippocampal neurons following traumatic brain injury is not associated with acute recovery of hippocampal functionHitting a moving target: Basic mechanisms of recovery from acquired developmental brain injury.Neuropathophysiology of Brain Injury.Mechanisms underlying the inability to induce area CA1 LTP in the mouse after traumatic brain injury.Single-neuron NMDA receptor phenotype influences neuronal rewiring and reintegration following traumatic injury.TrkB-enhancer facilitates functional recovery after traumatic brain injury.N-methyl-D-aspartate receptor subunit changes after traumatic injury to the developing brainAcute neuroprotection to pilocarpine-induced seizures is not sustained after traumatic brain injury in the developing rat.Dicyclomine, an M1 muscarinic antagonist, reduces biomarker levels, but not neuronal degeneration, in fluid percussion brain injury.The new pyridoindole antioxidant SMe1EC2 and its intervention in hypoxia/hypoglycemia-induced impairment of longterm potentiation in rat hippocampus.Therapeutic effects of ellagic acid on memory, hippocampus electrophysiology deficits, and elevated TNF-α level in brain due to experimental traumatic brain injury.Recovery of afferent function and synaptic strength in hippocampal CA1 following traumatic brain injury.Expression of protein phosphatase 2B (calcineurin) subunit A isoforms in rat hippocampus after traumatic brain injury.Reversal of trauma-induced amnesia in mice by a thrombin receptor antagonist.Metabolic, neurochemical, and histologic responses to vibrissa motor cortex stimulation after traumatic brain injury.In vitro mechanical strain trauma alters neuronal calcium responses: Implications for posttraumatic epilepsy.Converging early responses to brain injury pave the road to epileptogenesis.Phosphodiesterase-4 inhibition restored hippocampal long term potentiation after primary blast.
P2860
Q26752582-A0378DD8-3AFE-4A5F-B092-D15D06BA15B7Q28072353-284C8031-7A3C-4DBD-92BF-995CB4E024ECQ28385324-F6883AD0-E0AA-4DB0-96D7-12F82E15FA50Q33589580-89D8B948-F167-413C-9E82-F55B42408033Q34147858-4CB06F14-CC79-4AAE-961D-B48A4FEA984FQ34284469-48AEA4FC-7255-4CF8-8982-E7C5A66DF084Q34317749-5E0714BE-C68C-4A8B-B4BE-20293008895DQ34330618-1F8AE17C-10F0-482B-AB53-CDDEF1F6C7C0Q34777956-84775448-0C54-48B4-A637-D929F223C9E2Q34796693-5FA31E1E-E811-4B51-8CEB-569346BFEEF8Q34919457-38CBDD0A-A6B2-42C7-ADEF-D38CE52D6C36Q34980148-7B06AE66-5691-4518-8FF3-A6EDF55030C1Q35173229-7DCD2279-A2D7-42EA-A78E-6FAFF831982EQ35540623-9A18B092-5817-4F0F-A815-E9836172DC67Q35551108-C607D266-7ABF-4D45-8669-121AF4744DB4Q35885083-7FD86140-F38D-48E6-AF92-D7315DBDF9AEQ35972236-E3F81827-BFB3-436F-98DD-95AF83838905Q36446863-54B24A38-A9D1-4ED3-B666-9CC2AB8E000AQ36601929-4AD47F6D-3AE3-4034-8CA0-3172382F2D7EQ36622309-33D74A0B-CD38-4956-851F-57530E010765Q36835134-4E61E8E0-45AF-45D4-BD8F-64814D79EA81Q36848133-11268905-13B4-44FD-BE20-A1476EA4325DQ36964156-3A2D0AE0-0A86-4C36-AE5F-9D68B17D99D9Q37409435-0276BAB8-4195-4476-8497-F3F9B9344E6AQ37559491-6D7A2150-990E-41CB-BC7C-B17785B50527Q37633706-8541BE21-E524-4CEF-88B8-B4ED6B22F92CQ37650184-408E611C-B4A4-4C82-98D9-60B450F1F73BQ41480262-9DD5135E-A51C-45B3-8FCC-B2F2340411DCQ41857606-ABC9FDDD-23F4-4E8C-96F6-44A29E96E9CCQ41930078-6D420033-C07F-4F26-818D-EA98BBB81E44Q42182877-504EEB50-F99F-453B-A958-07630C7E7F59Q42211833-B366A8A5-3327-4AE8-8EAE-3A1B92C6F7C4Q42277857-2D6A6653-F695-4459-93CD-855B4F071E2AQ42912735-97C70806-E2AE-4BEC-918F-1FC236AFD299Q42928994-00896AAB-1FB4-46D3-B88B-6CDC0C178082Q44375033-ED4BE26C-F1CE-4669-AA5A-FF2041582E4AQ44539541-DD401EEE-224D-49ED-8212-802FF348ABC7Q47725049-4E5CF0E2-2C51-4ECC-AC8D-1386C399CCD8Q47941023-B0B7D9FA-CD20-4888-9134-EC1DC4BD7189Q48234922-353F7A13-0E3B-4799-9CD2-8A034746EFCB
P2860
Long-term potentiation deficits and excitability changes following traumatic brain injury.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh-hant
name
Long-term potentiation deficit ...... lowing traumatic brain injury.
@en
Long-term potentiation deficit ...... lowing traumatic brain injury.
@nl
type
label
Long-term potentiation deficit ...... lowing traumatic brain injury.
@en
Long-term potentiation deficit ...... lowing traumatic brain injury.
@nl
prefLabel
Long-term potentiation deficit ...... lowing traumatic brain injury.
@en
Long-term potentiation deficit ...... lowing traumatic brain injury.
@nl
P2093
P356
P1476
Long-term potentiation deficit ...... lowing traumatic brain injury.
@en
P2093
J T Povlishock
T M Reeves
P2888
P304
P356
10.1007/BF00241120
P577
1995-01-01T00:00:00Z
P6179
1034357936