Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
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Pharmacological manipulation of cell death: clinical applications in sight?Programmed necrosis in acute kidney injuryPreconditioning with triiodothyronine improves the clinical signs and acute tubular necrosis induced by ischemia/reperfusion in ratsReduced PARP1 as a Serum Biomarker for Graft Rejection in Kidney TransplantationMacrophages, dendritic cells, and kidney ischemia-reperfusion injury.Poly[ADP-ribose] polymerase-1 expression is related to cold ischemia, acute tubular necrosis, and delayed renal function in kidney transplantation.Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Targeted deletion of p53 in the proximal tubule prevents ischemic renal injuryp53 target Siva regulates apoptosis in ischemic kidneys.TIGAR regulates glycolysis in ischemic kidney proximal tubules.Anti-inflammatory effects of PJ34, a poly(ADP-ribose) polymerase inhibitor, in transient focal cerebral ischemia in miceThe role of necrotic cell death in the pathogenesis of immune mediated nephropathies.Peritubular ischemia contributes more to tubular damage than proteinuria in immune-mediated glomerulonephritis.Poly(ADP-ribose) polymerase-1 regulates the progression of autoimmune nephritis in males by inducing necrotic cell death and modulating inflammation.Therapeutic applications of PARP inhibitors: anticancer therapy and beyondRenal hypoxia and dysoxia after reperfusion of the ischemic kidney.Parp1 protects against Aag-dependent alkylation-induced nephrotoxicity in a sex-dependent mannerSildenafil ameliorates oxidative stress and DNA damage in the stenotic kidneys in mice with renovascular hypertension.The role of innate immunity in donor organ procurement.Role of poly (ADP-ribose)-polymerase and its signaling pathway with renin-angiotensin aldosterone system in renal diseases.Necroinflammation in Kidney Disease.Loss of poly(ADP-ribose) polymerase 1 attenuates renal fibrosis and inflammation during unilateral ureteral obstruction.MicroRNA-489 Induction by Hypoxia-Inducible Factor-1 Protects against Ischemic Kidney Injury.Inhibition of poly(adenosine diphosphate-ribose) polymerase attenuates ventilator-induced lung injury.TRPM2 mediates ischemic kidney injury and oxidant stress through RAC1.PARP-1 inhibits glycolysis in ischemic kidneys.PERP, a p53 proapoptotic target, mediates apoptotic cell death in renal ischemia.Cyclophilin D gene ablation protects mice from ischemic renal injury.PJ34, a poly adenosine diphosphate-ribose polymerase inhibitor, attenuates chromate-induced nephrotoxicity.Role of PARP activity in lung cancer-induced cachexia: Effects on muscle oxidative stress, proteolysis, anabolic markers, and phenotype.Potential role of poly(ADP-ribose) polymerase (PARP) activation in methotrexate-induced nephrotoxicity and tubular apoptosis.Immune mechanisms in the different phases of acute tubular necrosis
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P2860
Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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name
Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
@en
type
label
Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
@en
prefLabel
Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
@en
P2093
P2860
P1476
Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury.
@en
P2093
Babu J Padanilam
Jianfeng Zheng
Kishor Devalaraja-Narashimha
Kurinji Singaravelu
P2860
P304
P356
10.1152/AJPRENAL.00436.2003
P577
2004-10-19T00:00:00Z