Dopamine modulates excitability of basolateral amygdala neurons in vitro.
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Inhibitory networks of the amygdala for emotional memory.Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disordersMolecular and neuronal plasticity mechanisms in the amygdala-prefrontal cortical circuit: implications for opiate addiction memory formationBasolateral amygdala rapid glutamate release encodes an outcome-specific representation vital for reward-predictive cues to selectively invigorate reward-seeking actions.Dopamine-induced plasticity, phospholipase D (PLD) activity and cocaine-cue behavior depend on PLD-linked metabotropic glutamate receptors in amygdalaMechanisms of fear extinctionMechanisms to medicines: elucidating neural and molecular substrates of fear extinction to identify novel treatments for anxiety disordersDopamine receptor mechanisms mediate corticotropin-releasing factor-induced long-term potentiation in the rat amygdala following cocaine withdrawalDopamine modulates the excitability of projection neurons in the robust nucleus of the arcopallium in adult zebra finchesAffect-modulated startle: interactive influence of catechol-O-methyltransferase Val158Met genotype and childhood trauma.Amygdala Dopamine Receptors Are Required for the Destabilization of a Reconsolidating Appetitive Memory(1,2).Dopaminergic Modulation of Lateral Amygdala Neuronal Activity: Differential D1 and D2 Receptor Effects on Thalamic and Cortical Afferent Inputs.Fear signaling in the prelimbic-amygdala circuit: a computational modeling and recording study.Presynaptic facilitation of glutamate release in the basolateral amygdala: a mechanism for the anxiogenic and seizurogenic function of GluK1 receptors.Long-term memory for pavlovian fear conditioning requires dopamine in the nucleus accumbens and basolateral amygdala.Distribution of D1 and D5 dopamine receptors in the primate and rat basolateral amygdala.Dopamine is necessary for cue-dependent fear conditioning.Dopamine enhances fast excitatory synaptic transmission in the extended amygdala by a CRF-R1-dependent process.Dopamine modulation of emotional processing in cortical and subcortical neural circuits: evidence for a final common pathway in schizophrenia?Association between amygdala reactivity and a dopamine transporter gene polymorphism.Moderate Alcohol Exposure during the Rat Equivalent to the Third Trimester of Human Pregnancy Alters Regulation of GABAA Receptor-Mediated Synaptic Transmission by Dopamine in the Basolateral Amygdala.Regulation of Fear Extinction in the Basolateral Amygdala by Dopamine D2 Receptors Accompanied by Altered GluR1, GluR1-Ser845 and NR2B Levels.Memory enhancement induced by post-training intrabasolateral amygdala infusions of beta-adrenergic or muscarinic agonists requires activation of dopamine receptors: Involvement of right, but not left, basolateral amygdalaIntrinsic neuronal plasticity in the juxtacapsular nucleus of the bed nuclei of the stria terminalis (jcBNST)Protracted withdrawal from alcohol and drugs of abuse impairs long-term potentiation of intrinsic excitability in the juxtacapsular bed nucleus of the stria terminalis.Linking variability in brain chemistry and circuit function through multimodal human neuroimaging.Dopamine attenuates evoked inhibitory synaptic currents in central amygdala neurons.Early versus late-phase consolidation of opiate reward memories requires distinct molecular and temporal mechanisms in the amygdala-prefrontal cortical pathway.Dopamine D3-like receptors modulate anxiety-like behavior and regulate GABAergic transmission in the rat lateral/basolateral amygdala.Amygdala-prefrontal pathways and the dopamine system affect nociceptive responses in the prefrontal cortexStructure of distress call: implication for specificity and activation of dopaminergic system.Third trimester-equivalent ethanol exposure increases anxiety-like behavior and glutamatergic transmission in the basolateral amygdalaFunctional significance of central D1 receptors in cognition: beyond working memory.A Selective Role for Lmo4 in Cue-Reward Learning.Synaptic competition in the lateral amygdala and the stimulus specificity of conditioned fear: a biophysical modeling study.Attention-related Pearce-Kaye-Hall signals in basolateral amygdala require the midbrain dopaminergic system.The basolateral amygdala γ-aminobutyric acidergic system in health and disease.CRF2 Receptor Deficiency Eliminates the Long-Lasting Vulnerability of Motivational States Induced by Opiate WithdrawalDopaminergic innervation of interneurons in the rat basolateral amygdalaGoal-Directed Behavior and Instrumental Devaluation: A Neural System-Level Computational Model.
P2860
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P2860
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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name
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@en
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@nl
type
label
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@en
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@nl
prefLabel
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@en
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@nl
P2093
P2860
P356
P1476
Dopamine modulates excitability of basolateral amygdala neurons in vitro.
@en
P2093
German Barrionuevo
J Amiel Rosenkranz
Sven Kröner
P2860
P304
P356
10.1152/JN.00843.2004
P407
P577
2004-11-10T00:00:00Z