Insulin and contraction stimulate exocytosis, but increased AMP-activated protein kinase activity resulting from oxidative metabolism stress slows endocytosis of GLUT4 in cardiomyocytes.
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Translocation of the Na+/H+ exchanger 1 (NHE1) in cardiomyocyte responses to insulin and energy-status signallingClathrin-dependent and independent endocytosis of glucose transporter 4 (GLUT4) in myoblasts: regulation by mitochondrial uncouplingKinetic evidence for unique regulation of GLUT4 trafficking by insulin and AMP-activated protein kinase activators in L6 myotubesReciprocal regulation of endocytosis and metabolismOuabain attenuates cardiotoxicity induced by other cardiac steroids.Kinetic evidence that Glut4 follows different endocytic pathways than the receptors for transferrin and alpha2-macroglobulin.Subcellular trafficking of the substrate transporters GLUT4 and CD36 in cardiomyocytesPhysical developmental cues for the maturation of human pluripotent stem cell-derived cardiomyocytes.AMP-Activated Protein Kinase Regulates the Cell Surface Proteome and Integrin Membrane Traffic.Skeletal muscle glucose uptake during exercise: how is it regulated?Role of monosaccharide transport proteins in carbohydrate assimilation, distribution, metabolism, and homeostasis.Role of AMP-activated protein kinase in healthy and diseased hearts.E3 ubiquitin ligase, WWP1, interacts with AMPKα2 and down-regulates its expression in skeletal muscle C2C12 cells.Alterations in energy metabolism in cardiomyopathies.Contraction-stimulated glucose transport in rat skeletal muscle is sustained despite reversal of increased PAS-phosphorylation of AS160 and TBC1D1RLIP76 protein knockdown attenuates obesity due to a high-fat dietAberrant endoplasmic reticulum stress in vascular smooth muscle increases vascular contractility and blood pressure in mice deficient of AMP-activated protein kinase-α2 in vivo.Resistin, an adipocytokine, offers protection against acute myocardial infarction.A common trafficking route for GLUT4 in cardiomyocytes in response to insulin, contraction and energy-status signallingMetformin attenuates pressure overload-induced cardiac hypertrophy via AMPK activation.Measuring GLUT4 translocation in mature muscle fibers.Compensation: a contemporary regulatory machinery in cardiovascular diseases?AMP-activated protein kinase regulation and biological actions in the heart.AMPK: energy sensor and survival mechanism in the ischemic heart.Role of microRNA in metabolic shift during heart failure.Role of AMP-Activated Protein Kinase for Regulating Post-exercise Insulin Sensitivity.AMPK in Cardiovascular Diseases.Membrane Cholesterol in Skeletal Muscle: A Novel Player in Excitation-Contraction Coupling and Insulin Resistance.AMPK inactivation in mononuclear cells: a potential intracellular mechanism for exercise-induced immunosuppression.Regulation of AMP-activated protein kinase by cAMP in adipocytes: roles for phosphodiesterases, protein kinase B, protein kinase A, Epac and lipolysis.Kinetics of GLUT4 trafficking in rat and human skeletal muscle.Acute regulation of 5'-AMP-activated protein kinase by long-chain fatty acid, glucose and insulin in rat primary adipocytes.Shuttling glucose across brain microvessels, with a little help from GLUT1 and AMP kinase. Focus on "AMP kinase regulation of sugar transport in brain capillary endothelial cells during acute metabolic stress".AMP kinase activation and glut4 translocation in isolated cardiomyocytesHypoxia induces PGC-1α expression and mitochondrial biogenesis in the myocardium of TOF patients.Effects of AMPK activators on the sub-cellular distribution of fatty acid transporters CD36 and FABPpm.Myocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection.AMPK activation with AICAR provokes an acute fall in plasma [K+].Alpha-actinin-4 is selectively required for insulin-induced GLUT4 translocation.Muscle cell depolarization induces a gain in surface GLUT4 via reduced endocytosis independently of AMPK.
P2860
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P2860
Insulin and contraction stimulate exocytosis, but increased AMP-activated protein kinase activity resulting from oxidative metabolism stress slows endocytosis of GLUT4 in cardiomyocytes.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@en
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@nl
type
label
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@en
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@nl
prefLabel
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@en
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@nl
P2860
P356
P1476
Insulin and contraction stimul ...... is of GLUT4 in cardiomyocytes.
@en
P2093
P2860
P304
P356
10.1074/JBC.M410213200
P407
P577
2004-11-22T00:00:00Z