Paradoxical enhancement of active cochlear mechanics in long-term administration of salicylate.
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Too much of a good thing: long-term treatment with salicylate strengthens outer hair cell function but impairs auditory neural activityTinnitus: characteristics, causes, mechanisms, and treatmentsMeta-Analysis of Distortion Product Otoacoustic Emission Retest Variability for Serial Monitoring of Cochlear Function in Adults.Salicylate-induced cochlear impairments, cortical hyperactivity and re-tuning, and tinnitus.Salicylate-induced degeneration of cochlea spiral ganglion neurons-apoptosis signalingLong-term administration of salicylate enhances prestin expression in rat cochleaEffects of long-term salicylate administration on synaptic ultrastructure and metabolic activity in the rat CNS.Salicylate-induced changes in immediate-early genes in the hippocampal CA1 area.Animal models of subjective tinnitus.Expression of immediate-early genes in the inferior colliculus and auditory cortex in salicylate-induced tinnitus in ratPrestin up-regulation in chronic salicylate (aspirin) administration: an implication of functional dependence of prestin expression.Analgesic use and the risk of hearing loss in menIn vitro and in vivo models of drug ototoxicity: studying the mechanisms of a clinical problem.Understanding drug ototoxicity: molecular insights for prevention and clinical management.The complexity of age-related hearing impairment: contributing environmental and genetic factors.Salicylate selectively kills cochlear spiral ganglion neurons by paradoxically up-regulating superoxide.Long-term Administration of Salicylate-induced Changes in BDNF Expression and CREB Phosphorylation in the Auditory Cortex of Rats.Expression of immediate-early genes in the dorsal cochlear nucleus in salicylate-induced tinnitus.Salicylate-Induced Hearing Loss Trigger Structural Synaptic Modifications in the Ventral Cochlear Nucleus of Rats via Medial Olivocochlear (MOC) Feedback Circuit.Effects of salicylate on the inflammatory genes expression and synaptic ultrastructure in the cochlear nucleus of rats.Effects of whole body vibration on outer hair cells' hearing response to distortion product otoacoustic emissions.Overall versus individual changes for otoacoustic emissions and audiometry in a noise-exposed cohort.Salicylate initiates apoptosis in the spiral ganglion neuron of guinea pig cochlea by activating caspase-3.Paradoxical long-term enhancement of distortion product otoacoustic emission amplitude after repeated exposure to moderate level, wide band noise in awake guinea pigs.
P2860
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P2860
Paradoxical enhancement of active cochlear mechanics in long-term administration of salicylate.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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name
Paradoxical enhancement of act ...... administration of salicylate.
@en
Paradoxical enhancement of act ...... administration of salicylate.
@nl
type
label
Paradoxical enhancement of act ...... administration of salicylate.
@en
Paradoxical enhancement of act ...... administration of salicylate.
@nl
prefLabel
Paradoxical enhancement of act ...... administration of salicylate.
@en
Paradoxical enhancement of act ...... administration of salicylate.
@nl
P2093
P2860
P356
P1476
Paradoxical enhancement of act ...... administration of salicylate.
@en
P2093
Hong-Bo Zhao
Raleigh O Jones
Yanyun Luo
Zhanyuan Wu
Zhezhang Tao
Zhi-Wu Huang
P2860
P304
P356
10.1152/JN.00959.2004
P407
P577
2004-12-08T00:00:00Z