Effects of infliximab on apoptosis and reverse signaling of monocytes from healthy individuals and patients with Crohn's disease.
about
Role of Tumor Necrosis Factor Superfamily in Neuroinflammation and AutoimmunityUse of the tumor necrosis factor-blockers for Crohn's diseaseMolecular mechanism of action of anti-tumor necrosis factor antibodies in inflammatory bowel diseasesCD25 and TNF receptor II reflect early primary response to infliximab therapy in patients with ulcerative colitisCurrent status of monoclonal antibody therapy for the treatment of inflammatory bowel disease.Characterization of golimumab, a human monoclonal antibody specific for human tumor necrosis factor α.Development of a Recombinant Xenogeneic Tumor Necrosis Factor Alpha Protein Vaccine To Protect Mice from Experimental Colitis.TNFalpha blockade in human diseases: mechanisms and future directions.Emerging drugs to treat Crohn's disease.Exposure to nuclear antigens contributes to the induction of humoral autoimmunity during tumour necrosis factor alpha blockade.Importance of reverse signaling of the TNF superfamily in immune regulation.Mechanisms of action of anti-tumor necrosis factor α agents in Crohn's disease.New Insights into the Mechanisms of Action of Anti-Tumor Necrosis Factor-α Monoclonal Antibodies in Inflammatory Bowel Disease.A real-world risk analysis of biological treatment (adalimumab and etanercept) in a country with a high prevalence of tuberculosis and chronic liver disease: a nationwide population-based study.The anti-TNF-α antibody infliximab indirectly regulates PECAM-1 gene expression in two models of in vitro blood cell activation.Comparable Immune Function Inhibition by the Infliximab Biosimilar CT-P13: Implications for Treatment of Inflammatory Bowel Disease.Reverse signalling of membrane-integrated tumour necrosis factor differentially regulates alloresponses of CD4+ and CD8+ T cells against human microvascular endothelial cells.Transmembrane TNF-α promotes activation-induced cell death by forward and reverse signalingNon-response to infliximab may be due to innate neutralizing anti-tumour necrosis factor-alpha antibodies.Infliximab inhibits activation and effector functions of peripheral blood T cells in vitro from patients with clinically active ulcerative colitis.Structural Biology of the TNFα Antagonists Used in the Treatment of Rheumatoid Arthritis.Infliximab Selectively Modulates the Circulating Blood Monocyte Repertoire in Crohn's Disease.
P2860
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P2860
Effects of infliximab on apoptosis and reverse signaling of monocytes from healthy individuals and patients with Crohn's disease.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
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2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
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2004年學術文章
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2004年學術文章
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name
Effects of infliximab on apopt ...... patients with Crohn's disease.
@en
Effects of infliximab on apopt ...... patients with Crohn's disease.
@nl
type
label
Effects of infliximab on apopt ...... patients with Crohn's disease.
@en
Effects of infliximab on apopt ...... patients with Crohn's disease.
@nl
prefLabel
Effects of infliximab on apopt ...... patients with Crohn's disease.
@en
Effects of infliximab on apopt ...... patients with Crohn's disease.
@nl
P2093
P2860
P1476
Effects of infliximab on apopt ...... patients with Crohn's disease
@en
P2093
Fredric Daum
Jack Silver
James Markowitz
Mihaela Ringheanu
Xingyu Lin
P2860
P304
P356
10.1097/00054725-200411000-00015
P577
2004-11-01T00:00:00Z