Reactive oxygen species reduce myofibrillar Ca2+ sensitivity in fatiguing mouse skeletal muscle at 37 degrees C.
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The Measurement of Reversible Redox Dependent Post-translational Modifications and Their Regulation of Mitochondrial and Skeletal Muscle FunctionPotential molecular mechanisms underlying muscle fatigue mediated by reactive oxygen and nitrogen species0.9% saline is neither normal nor physiologicalDiaphragm atrophy and contractile dysfunction in a murine model of pulmonary hypertensionPeroxynitrite inhibits myofibrillar protein function in an in vitro assay of motilityThe excitation-contraction coupling mechanism in skeletal muscle.Exercise-induced oxidative stress: cellular mechanisms and impact on muscle force productionSphingomyelinase stimulates oxidant signaling to weaken skeletal muscle and promote fatigue.Angiotensin II modulates mouse skeletal muscle resting conductance to chloride and potassium ions and calcium homeostasis via the AT1 receptor and NADPH oxidaseRecent insights into muscle fatigue at the cross-bridge level.Mitochondrial redox potential during contraction in single intact muscle fibers.Muscle-derived ROS and thiol regulation in muscle fatigue.Sphingomyelinase promotes oxidant production and skeletal muscle contractile dysfunction through activation of NADPH oxidase.Properties of single FDB fibers following a collagenase digestion for studying contractility, fatigue, and pCa-sarcomere shortening relationship.A tissue-engineered muscle repair construct for functional restoration of an irrecoverable muscle injury in a murine modelChemotherapy-induced weakness and fatigue in skeletal muscle: the role of oxidative stress.Differential involvement of various sources of reactive oxygen species in thyroxin-induced hemodynamic changes and contractile dysfunction of the heart and diaphragm muscles.Antioxidants and Skeletal Muscle Performance: "Common Knowledge" vs. Experimental Evidence.Effects of short-term antioxidant supplementation on oxidative stress and exercise performance in the heat and the cold.A new therapeutic effect of simvastatin revealed by functional improvement in muscular dystrophy.Mechanisms of stretch-induced muscle damage in normal and dystrophic muscle: role of ionic changes.Conditional knockout of Mn-SOD targeted to type IIB skeletal muscle fibers increases oxidative stress and is sufficient to alter aerobic exercise capacity.Reactive oxygen species: impact on skeletal muscle.Acute effects of reactive oxygen and nitrogen species on the contractile function of skeletal muscle.Effects of Ionizing Irradiation on Mouse Diaphragmatic Skeletal Muscle.Mechanisms Explaining Muscle Fatigue and Muscle Pain in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): a Review of Recent Findings.High temperature does not alter fatigability in intact mouse skeletal muscle fibres.Muscle fatigue examined at different temperatures in experiments on intact mammalian (rat) muscle fibers.Aerobic exercise training improves Ca2+ handling and redox status of skeletal muscle in mice.Influence of dietary nitrate supplementation on human skeletal muscle metabolism and force production during maximum voluntary contractions.Sequential effects of GSNO and H2O2 on the Ca2+ sensitivity of the contractile apparatus of fast- and slow-twitch skeletal muscle fibers from the rat.Time to fatigue is increased in mouse muscle at 37 degrees C; the role of iron and reactive oxygen species.Po2 cycling protects diaphragm function during reoxygenation via ROS, Akt, ERK, and mitochondrial channels.Iron injections in mice increase skeletal muscle iron content, induce oxidative stress and reduce exercise performance.Mechanical isolation, and measurement of force and myoplasmic free [Ca2+] in fully intact single skeletal muscle fibers.L-carnitine supplementation attenuates intermittent hypoxia-induced oxidative stress and delays muscle fatigue in rats.Effects of N-acetylcysteine on isolated mouse skeletal muscle: contractile properties, temperature dependence, and metabolism.Hydroxyl radical and glutathione interactions alter calcium sensitivity and maximum force of the contractile apparatus in rat skeletal muscle fibres.The activity-induced reduction of myofibrillar Ca2+ sensitivity in mouse skeletal muscle is reversed by dithiothreitol.Oxidation of myosin heavy chain and reduction in force production in hyperthyroid rat soleus.
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P2860
Reactive oxygen species reduce myofibrillar Ca2+ sensitivity in fatiguing mouse skeletal muscle at 37 degrees C.
description
2005 nî lūn-bûn
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2005年の論文
@ja
2005年学术文章
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2005年学术文章
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2005年学术文章
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name
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@en
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@nl
type
label
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@en
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@nl
prefLabel
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@en
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@nl
P2860
P1476
Reactive oxygen species reduce ...... eletal muscle at 37 degrees C.
@en
P2093
Terence R Moopanar
P2860
P304
P356
10.1113/JPHYSIOL.2005.083519
P407
P577
2005-02-17T00:00:00Z