A combination drug therapy improves cognition and reverses gene expression changes in a mouse model of Huntington's disease.
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Mouse models of polyglutamine diseases in therapeutic approaches: review and data table. Part II.A novel RING finger protein, Znf179, modulates cell cycle exit and neuronal differentiation of P19 embryonal carcinoma cellsMouse models of Huntington's disease and methodological considerations for therapeutic trials.Direct evidence of progressive cardiac dysfunction in a transgenic mouse model of Huntington's disease.Cholinergic neuronal defect without cell loss in Huntington's disease.CalDAG-GEFI down-regulation in the striatum as a neuroprotective change in Huntington's disease.Cystamine and intrabody co-treatment confers additional benefits in a fly model of Huntington's disease.The emerging role of guanine nucleotide exchange factors in ALS and other neurodegenerative diseases.A ketogenic diet delays weight loss and does not impair working memory or motor function in the R6/2 1J mouse model of Huntington's diseaseTranscription, epigenetics and ameliorative strategies in Huntington's Disease: a genome-wide perspectiveComponents of the endocannabinoid and dopamine systems are dysregulated in Huntington's disease: analysis of publicly available microarray datasets.Reductions in behavioral deficits and neuropathology in the R6/2 mouse model of Huntington's disease following transplantation of bone-marrow-derived mesenchymal stem cells is dependent on passage number.Similar striatal gene expression profiles in the striatum of the YAC128 and HdhQ150 mouse models of Huntington's disease are not reflected in mutant Huntingtin inclusion prevalence.Transcriptional signatures in Huntington's disease.Analysis of the interaction between Zinc finger protein 179 (Znf179) and promyelocytic leukemia zinc finger (Plzf).Striatal Neuropeptides Enhance Selection and Rejection of Sequential Actions.Brain gene expression correlates with changes in behavior in the R6/1 mouse model of Huntington's disease.Important Roles of Ring Finger Protein 112 in Embryonic Vascular Development and Brain Functions.Metabolic and transcriptomic analysis of Huntington's disease model reveal changes in intracellular glucose levels and related genesModeling cholesterol metabolism by gene expression profiling in the hippocampus.Cognitive Disorders: Impairment, Aging, and Dementia
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P2860
A combination drug therapy improves cognition and reverses gene expression changes in a mouse model of Huntington's disease.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
A combination drug therapy imp ...... model of Huntington's disease.
@en
A combination drug therapy imp ...... model of Huntington's disease.
@nl
type
label
A combination drug therapy imp ...... model of Huntington's disease.
@en
A combination drug therapy imp ...... model of Huntington's disease.
@nl
prefLabel
A combination drug therapy imp ...... model of Huntington's disease.
@en
A combination drug therapy imp ...... model of Huntington's disease.
@nl
P2093
P50
P1476
A combination drug therapy imp ...... model of Huntington's disease
@en
P2093
Amanda J Redfern
Angela K Hodges
Lesley Jones
Paul D Lewis
P304
P356
10.1111/J.1460-9568.2005.03895.X
P407
P577
2005-02-01T00:00:00Z