The calcium-binding protein parvalbumin modulates the firing 1 properties of the reticular thalamic nucleus bursting neurons.
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The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophreniaLack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities.Mice lacking the transcriptional coactivator PGC-1α exhibit alterations in inhibitory synaptic transmission in the motor cortex.Neurovascular EGFL7 regulates adult neurogenesis in the subventricular zone and thereby affects olfactory perception.Reduction in parvalbumin expression not loss of the parvalbumin-expressing GABA interneuron subpopulation in genetic parvalbumin and shank mouse models of autismGABAergic Function as a Limiting Factor for Prefrontal Maturation during Adolescence.Notch1 activity in the olfactory bulb is odour-dependent and contributes to olfactory behaviour.Cerebellins are differentially expressed in selective subsets of neurons throughout the brain.Schizophrenia: from neurophysiological abnormalities to clinical symptoms.
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The calcium-binding protein parvalbumin modulates the firing 1 properties of the reticular thalamic nucleus bursting neurons.
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name
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@en
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@nl
type
label
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@en
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@nl
prefLabel
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@en
The calcium-binding protein pa ...... amic nucleus bursting neurons.
@nl
P2860
P50
P356
P1476
The calcium-binding protein pa ...... lamic nucleus bursting neurons
@en
P2093
Lavinia Albéri
Robert Kretz
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P304
P356
10.1152/JN.00375.2012
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P577
2013-03-13T00:00:00Z