Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis. - Wikidata - wikimedia - TriplyDB

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

http://www.wikidata.org/entity/Q45331115

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Role of non-neuronal cells in body weight and appetite control Role of transient receptor potential ankyrin 1 channels in Alzheimer's disease Bexarotene reduces blood-brain barrier permeability in cerebral ischemia-reperfusion injured rats 3,4-oxo-isopropylidene-shikimic acid inhibits cerebral ischemia-induced oxidative stress and neuronal apoptosis in rats.Cerebrospinal fluid apolipoprotein E and phospholipid transfer protein activity are reduced in multiple sclerosis; relationships with the brain MRI and CSF lipid variables.An integrative genome-wide transcriptome reveals that candesartan is neuroprotective and a candidate therapeutic for Alzheimer's disease Apolipoprotein E-Mimetic COG1410 Reduces Acute Vasogenic Edema following Traumatic Brain Injury.ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells.Morphine Protects Spinal Cord Astrocytes from Glutamate-Induced Apoptosis via Reducing Endoplasmic Reticulum Stress.Potential implications of Apolipoprotein E in early brain injury after experimental subarachnoid hemorrhage: Involvement in the modulation of blood-brain barrier integrity.Gene expression related to oxidative stress in the heart of mice after intestinal ischemia.ApoE Influences the Blood-Brain Barrier Through the NF-κB/MMP-9 Pathway After Traumatic Brain Injury Thirty Minutes of Hypobaric Hypoxia Provokes Alterations of Immune Response, Haemostasis, and Metabolism Proteins in Human Serum Apolipoprotein E Genotype Affects Size of ApoE Complexes in Cerebrospinal Fluid.Orexin-A promotes Glu uptake by OX1R/PKCα/ERK1/2/GLT-1 pathway in astrocytes and protects co-cultured astrocytes and neurons against apoptosis in anoxia/hypoglycemic injury in vitro.

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Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

http://www.wikidata.org/entity/Q45331115

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-my

2012年学术文章

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2012年學術文章

@yue

2012年學術文章

@zh-hant

name

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@en

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@nl

type

label

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@en

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@nl

prefLabel

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@en

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@nl

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P1476

Apolipoprotein E protects astrocytes from hypoxia and glutamate-induced apoptosis.

@en

P2093

Chun Zeng

http://www.w3.org/2001/XMLSchema#string

Haitao Wu

http://www.w3.org/2001/XMLSchema#string

Shuai Zhou

http://www.w3.org/2001/XMLSchema#string

Shuang Tang

http://www.w3.org/2001/XMLSchema#string

Xiaochuan Sun

http://www.w3.org/2001/XMLSchema#string

Xuehua Xiong

http://www.w3.org/2001/XMLSchema#string

Zhaohua Tang

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P2860

Apolipoprotein E and its receptors in Alzheimer's disease: pathways, pathogenesis and therapy

Non-cell autonomous effect of glia on motor neurons in an embryonic stem cell-based ALS model

The protective effect of tetramethylpyrazine on cartilage explants and chondrocytes.

Glycosylation and sialylation of macrophage-derived human apolipoprotein E analyzed by SDS-PAGE and mass spectrometry: evidence for a novel site of glycosylation on Ser290.

Astrocytic beta2-adrenergic receptors and multiple sclerosis.

Astrocyte mitochondrial mechanisms of ischemic brain injury and neuroprotection.

Glutamate-mediated glial injury: mechanisms and clinical importance.

Comparison of alterations in amino acids content in cultured astrocytes or neurons exposed to methylmercury separately or in co-culture

Astrocyte-neuron interactions in neurological disorders.

The release and uptake of excitatory amino acids.

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254-258

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2

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