Accumulation of nuclear and mitochondrial DNA damage in the frontal cortex cells of patients with HIV-associated neurocognitive disorders.
about
PACAP27 is protective against tat-induced neurotoxicityRoles and functions of HIV-1 Tat protein in the CNS: an overviewUntangling the Gordian knot of HIV, stress, and cognitive impairmentOxidative Stress during HIV Infection: Mechanisms and ConsequencesRegional brain distribution of translocator protein using [(11)C]DPA-713 PET in individuals infected with HIV.Host genetic factors predisposing to HIV-associated neurocognitive disorder.Oxidative stress, mitochondrial damage and neurodegenerative diseasesHeme oxygenase-1 dysregulation in the brain: implications for HIV-associated neurocognitive disorders.Circulating brain microvascular endothelial cells (cBMECs) as potential biomarkers of the blood-brain barrier disorders caused by microbial and non-microbial factors.HIV effects on age-associated neurocognitive dysfunction: premature cognitive aging or neurodegenerative disease?MSM ameliorates HIV-1 Tat induced neuronal oxidative stress via rebalance of the glutathione cycleMitochondrial DNA D-loop AG/TC transition mutation in cortical neurons of mice after long-term exposure to nucleoside analogues.Mitochondrial DNA Haplogroups and Neurocognitive Impairment During HIV Infection.HIV-1 gp120 induces type-1 programmed cell death through ER stress employing IRE1α, JNK and AP-1 pathway.Dimethyl fumarate modulation of immune and antioxidant responses: application to HIV therapy.Development of TIMP1 magnetic nanoformulation for regulation of synaptic plasticity in HIV-1 infection.Different levels of HIV DNA copy numbers in cerebrospinal fluid cellular subsets.Biological and physiological role of reactive oxygen species--the good, the bad and the ugly.Interventions for Neurocognitive Dysfunction.The potential toxicological insights about the anti-HIV drug azidothymidine-derived monoselenides in human leukocytes: Toxicological insights of new selenium-azidothymidine analogs.Childhood maltreatment and HIV-associated neurocognitive disorders share similar pathophysiology: a potential sensitisation mechanism?Human Immunodeficiency Virus Promotes Mitochondrial Toxicity.DRAM Is Involved in Regulating Nucleoside Analog-Induced Neuronal Autophagy in a p53-Independent Manner.Long-term exposure of mice to nucleoside analogues disrupts mitochondrial DNA maintenance in cortical neurons.Discordant patterns of tissue-specific genetic characteristics in the HIV-1 env gene from HIV-associated neurocognitive disorder (HAND) and non-HAND patients.High levels of divergent HIV-1 quasispecies in patients with neurological opportunistic infections in China.Neural response to working memory demand predicts neurocognitive deficits in HIV.
P2860
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P2860
Accumulation of nuclear and mitochondrial DNA damage in the frontal cortex cells of patients with HIV-associated neurocognitive disorders.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@en
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@nl
type
label
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@en
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@nl
prefLabel
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@en
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@nl
P2093
P1433
P1476
Accumulation of nuclear and mi ...... ated neurocognitive disorders.
@en
P2093
Daojie Liu
Honghai Zhang
Hongjun Li
Meixia Wang
Yulin Zhang
P356
10.1016/J.BRAINRES.2012.04.001
P407
P577
2012-04-11T00:00:00Z