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Loss of translation: a stealth weapon against pathogens?

Loss of translation: a stealth weapon against pathogens?

http://www.wikidata.org/entity/Q45419450

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Protein synthesis inhibition and GADD34 control IFN-β heterogeneous expression in response to dsRNA.

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P2860

Loss of translation: a stealth weapon against pathogens?

http://www.wikidata.org/entity/Q45419450

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Loss of translation: a stealth weapon against pathogens?

@en

Loss of translation: a stealth weapon against pathogens?

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type

label

Loss of translation: a stealth weapon against pathogens?

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Loss of translation: a stealth weapon against pathogens?

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prefLabel

Loss of translation: a stealth weapon against pathogens?

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Loss of translation: a stealth weapon against pathogens?

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P1433

Nature Immunology

P1476

Loss of translation: a stealth weapon against pathogens?

@en

P2093

Evelina Gatti

http://www.w3.org/2001/XMLSchema#string

Philippe Pierre

http://www.w3.org/2001/XMLSchema#string

P2860

Translational control of the activation of transcription factor NF-κB and production of type I interferon by phosphorylation of the translation factor eIF4E.

Secreted bacterial effectors that inhibit host protein synthesis are critical for induction of the innate immune response to virulent Legionella pneumophila.

Host innate immune receptors and beyond: making sense of microbial infections.

TOR in the immune system.

Regulation of translation is required for dendritic cell function and survival during activation.

Immunity and the regulation of protein synthesis: surprising connections.

Ribosomal protein mRNAs are translationally-regulated during human dendritic cells activation by LPS.

Patterns of pathogenesis: discrimination of pathogenic and nonpathogenic microbes by the innate immune system.

Host translation at the nexus of infection and immunity.

Translation inhibition and metabolic stress pathways in the host response to bacterial pathogens.

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1203-1205

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scholarly article

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10.1038/NI.2759

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P407

P433

12

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14

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Philippe Pierre

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2013-12-01T00:00:00Z

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24240150

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