Inhibition of tumor necrosis factor reduces the severity of virus-specific lung immunopathology. - Wikidata - wikimedia - TriplyDB

Inhibition of tumor necrosis factor reduces the severity of virus-specific lung immunopathology.

Inhibition of tumor necrosis factor reduces the severity of virus-specific lung immunopathology.

http://www.wikidata.org/entity/Q45735792

about

Pathology, molecular biology, and pathogenesis of avian influenza A (H5N1) infection in humans Interleukin-1 is responsible for acute lung immunopathology but increases survival of respiratory influenza virus infection A critical role for OX40 in T cell-mediated immunopathology during lung viral infection A role for airway remodeling during respiratory syncytial virus infection.Anti-TNF therapy: past, present and future Evolution of parasite virulence when host responses cause disease Double-stranded RNA-activated protein kinase regulates early innate immune responses during respiratory syncytial virus infection Global host immune response: pathogenesis and transcriptional profiling of type A influenza viruses expressing the hemagglutinin and neuraminidase genes from the 1918 pandemic virus.Novel strategies for prevention and treatment of influenza.The effector T cell response to influenza infection Respiratory infections: do we ever recover?Protective memory responses are modulated by priming events prior to challenge.Collectins, H-ficolin and LL-37 reduce influence viral replication in human monocytes and modulate virus-induced cytokine production.Anti-tumor necrosis factor therapy and influenza: keeping it in perspective.Influenza-infected neutrophils within the infected lungs act as antigen presenting cells for anti-viral CD8(+) T cells Harnessing alveolar macrophages for sustained mucosal T-cell recall confers long-term protection to mice against lethal influenza challenge without clinical disease.Antiviral CD8+ T cell effector activities in situ are regulated by target cell type Fas ligand is required for the development of respiratory syncytial virus vaccine-enhanced disease Prioritizing genes responsible for host resistance to influenza using network approaches Sub-nucleocapsid nanoparticles: a nasal vaccine against respiratory syncytial virus.The chemokine MIP1alpha/CCL3 determines pathology in primary RSV infection by regulating the balance of T cell populations in the murine lung.Respiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.Soluble, but not transmembrane, TNF-α is required during influenza infection to limit the magnitude of immune responses and the extent of immunopathology The meteorology of cytokine storms, and the clinical usefulness of this knowledge Innate immunity to influenza virus infection.The migration of T cells in response to influenza virus is altered in neonatal mice.Production and differentiation of myeloid cells driven by proinflammatory cytokines in response to acute pneumovirus infection in mice Transgenic expression in mouse lung reveals distinct biological roles for the adenovirus type 5 E1A 243- and 289-amino-acid proteins The airway epithelium: soldier in the fight against respiratory viruses Infection-triggered regulatory mechanisms override the role of STAT 4 in control of the immune response to influenza virus antigens.Differentiation-dependent differences in murine T cell susceptibility to negative regulation by the lung Exposure of neonates to respiratory syncytial virus is critical in determining subsequent airway response in adults.ADAM17-mediated processing of TNF-α expressed by antiviral effector CD8+ T cells is required for severe T-cell-mediated lung injury.Monoclonal antibody specific to HA2 glycopeptide protects mice from H3N2 influenza virus infection Bronchoalveolar lavage cellularity in infants with severe respiratory syncytial virus bronchiolitis.Influenza virus pathophysiology and brain invasion in mice with functional and dysfunctional Mx1 genes.Viral infection of the lungs through the eye.Transcriptomic and Epigenetic Profiling of the Lung of Influenza-Infected Pigs: A Comparison of Different Birth Weight and Susceptibility Groups.Role of host cytokine responses in the pathogenesis of avian H5N1 influenza viruses in mice.Into the eye of the cytokine storm.

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P2860

Inhibition of tumor necrosis factor reduces the severity of virus-specific lung immunopathology.

http://www.wikidata.org/entity/Q45735792

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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2001年学术文章

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Inhibition of tumor necrosis f ...... specific lung immunopathology.

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Inhibition of tumor necrosis f ...... specific lung immunopathology.

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Inhibition of tumor necrosis f ...... specific lung immunopathology.

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Inhibition of tumor necrosis f ...... specific lung immunopathology.

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European Journal of Immunology

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Openshaw PJ

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Pennycook A

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P2860

Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice

Virus-specific CD8+ T lymphocytes downregulate T helper cell type 2 cytokine secretion and pulmonary eosinophilia during experimental murine respiratory syncytial virus infection.

Local and systemic cytokine responses during experimental human influenza A virus infection. Relation to symptom formation and host defense

On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease.

Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment.

Recent developments in the biology of respiratory syncytial virus: are vaccines and new treatments just round the corner?

Respiratory syncytial virus infection.

Tumor necrosis factor: a master-regulator of leukocyte movement.

Cytotoxic T cells clear virus but augment lung pathology in mice infected with respiratory syncytial virus

Distinct types of lung disease caused by functional subsets of antiviral T cells.

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