RAD51 deficiency disrupts the corticospinal lateralization of motor control.
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A Dominant Mutation in Human RAD51 Reveals Its Function in DNA Interstrand Crosslink Repair Independent of Homologous RecombinationNon cell-autonomous role of DCC in the guidance of the corticospinal tract at the midline.Congenital Mirror Movements Due to RAD51: Cosegregation with a Nonsense Mutation in a Norwegian Pedigree and Review of the LiteratureSelective chromatid segregation mechanism invoked for the human congenital mirror hand movement disorder development by RAD51 mutations: a hypothesisA novel role for the DNA repair gene Rad51 in Netrin-1 signalling.Whole-exome sequencing of 228 patients with sporadic Parkinson's diseaseOne hand clapping: lateralization of motor control.The corticospinal tract: Evolution, development, and human disorders.Congenital mirror movements: mutational analysis of RAD51 and DCC in 26 cases.Identification of a homozygous splice site mutation in the dynein axonemal light chain 4 gene on 22q13.1 in a large consanguineous family from Pakistan with congenital mirror movement disorder.Congenital mirror movements: lack of decussation of pyramids.Reply: Congenital mirror movements: lack of decussation of pyramids Mirror movement: from physiopathology to treatment perspectives.Mutations in DCC cause isolated agenesis of the corpus callosum with incomplete penetrance.Mutations in the netrin-1 gene cause congenital mirror movements.Mirror Motor Activity During Right-Hand Contractions and Its Relation to White Matter in the Posterior Midbody of the Corpus Callosum.Structural Neural Correlates of Physiological Mirror Activity During Isometric Contractions of Non-Dominant Hand Muscles.Abnormal subcortical activity in congenital mirror movement disorder with RAD51 mutation
P2860
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P2860
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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name
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@en
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@nl
type
label
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@en
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@nl
prefLabel
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@en
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@nl
P2093
P2860
P50
P356
P1433
P1476
RAD51 deficiency disrupts the corticospinal lateralization of motor control.
@en
P2093
Benoît Schmitt
Christel Depienne
Christine Delmaire
Constance Flamand-Roze
Cyril Poupon
Cécile Gallea
Cécile Hubsch
Emmanuel Roze
Eric Bardinet
Eric Bertasi
P2860
P304
P356
10.1093/BRAIN/AWT258
P407
P577
2013-09-20T00:00:00Z